Literature DB >> 19888930

White matter lesion load increases the risk of low CSF Aβ42 in apolipoprotein E-ɛ4 carriers attending a memory clinic.

Vidar Stenset1, Dag Hofoss, Lisbeth Johnsen, Audun Elnaes Berstad, Anne Negaard, Anders Skinningsrud, Leif Gjerstad, Tormod Fladby.   

Abstract

BACKGROUND: White matter lesions (WMLs) are age-related manifestations of ischemic cerebrovascular disease and increase the risk for Alzheimer's disease (AD). The apolipoprotein E (ApoE) ɛ4 allele is a risk factor for late onset AD and has been related to low cerebrospinal fluid (CSF) Aβ42 levels and to cerebrovascular disease. The present study analyzed the relationship between WMLs, ApoE-ɛ4 genotype, and low CSF Aβ42.
METHODS: A total of 235 memory clinic attenders were stratified in 3 groups according to WML load. WMLs were rated on axial T2 magnetic resonance imaging images. Group 1 had no or only small amounts of periventricular (PV) or subcortical (SC) WMLs, WML group 2 had high amounts of PV WMLs and low amounts of SC WMLs, and WML group 3 had high amounts of both PV and SC WMLs. In each WML group, ApoE-ɛ4 genotype was used in logistic regression as a predictor for low CSF Aβ42 (cutoff≤450 ng/L).
RESULTS: The odds ratio (OR) of having low CSF Aβ42 was significantly increased in the presence of ApoE-ɛ4 only in WML group 3 (OR 3.69, P=.009).
CONCLUSION: A high WML load may interact with the ApoE-ɛ4 genotype and increase the risk for reduced CSF Aβ42 in patients attending a memory clinic.
Copyright © 2009 by the American Society of Neuroimaging.

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Year:  2011        PMID: 19888930     DOI: 10.1111/j.1552-6569.2009.00444.x

Source DB:  PubMed          Journal:  J Neuroimaging        ISSN: 1051-2284            Impact factor:   2.486


  8 in total

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5.  White matter hyperintensities are more highly associated with preclinical Alzheimer's disease than imaging and cognitive markers of neurodegeneration.

Authors:  Benjamin M Kandel; Brian B Avants; James C Gee; Corey T McMillan; Guray Erus; Jimit Doshi; Christos Davatzikos; David A Wolk
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6.  Amyloid Dysmetabolism Relates to Reduced Glucose Uptake in White Matter Hyperintensities.

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7.  Ventricular and Periventricular Anomalies in the Aging and Cognitively Impaired Brain.

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8.  Low Cerebrospinal Fluid Aβ42 and Aβ40 are Related to White Matter Lesions in Cognitively Normal Elderly.

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  8 in total

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