Literature DB >> 19887196

Thrombospondin-1-induced vascular smooth muscle cell migration is dependent on the hyaluronic acid receptor CD44.

Kristopher G Maier1, Benjamin Sadowitz, Sarah Cullen, Xuan Han, Vivian Gahtan.   

Abstract

BACKGROUND: Thrombospondin-1 (TSP-1) induces vascular smooth muscle cell (VSMC) migration after arterial injury. TSP-1 up-regulates hyaluronic acid (HyA)-inducing genes in VSMCs. HyA also induces VSMC migration. Our hypothesis was that TSP-1-induced VSMC migration is dependent on the CD44 receptor, and that HyA and TSP-1 share migratory signaling pathways.
METHODS: VSMC migration was assessed using TSP-1, HyA, or serum-free medium as chemoattractants. VSMCs were treated with inhibitors to CD44, Ras, phosphatidylinositol-3 kinase, Raf-1 kinase, or c-SRC. TSP-1- and HyA-induced epidermal growth factor receptor (EGFR) activity was determined by enzyme-linked immunosorbent assay. Comparisons were made by the Student t test and a P value less than .05 was considered significant.
RESULTS: Inhibiting CD44 reduced TSP-1- and HyA-induced migration. Phosphatidylinositol-3 kinase and c-SRC inhibitors prevented TSP-1- and HyA-induced migration, whereas Ras and Raf-1 kinase inhibitors only affected TSP-1. TSP-1 and HyA activate the EGFR.
CONCLUSIONS: TSP-1- and HYA-induced migration share some of the same signaling pathways and the EGFR/CD44 receptors may be a common link.

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Year:  2009        PMID: 19887196     DOI: 10.1016/j.amjsurg.2009.07.018

Source DB:  PubMed          Journal:  Am J Surg        ISSN: 0002-9610            Impact factor:   2.565


  7 in total

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2.  Plasma rich in growth factors (PRGF-Endoret) stimulates tendon and synovial fibroblasts migration and improves the biological properties of hyaluronic acid.

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3.  Thrombospondin-1 differentially regulates microRNAs in vascular smooth muscle cells.

Authors:  Kristopher G Maier; Brian Ruhle; Jeffrey J Stein; Karen L Gentile; Frank A Middleton; Vivian Gahtan
Journal:  Mol Cell Biochem       Date:  2016-01-04       Impact factor: 3.396

4.  Thrombospondin-1 null mice are resistant to hypoxia-induced pulmonary hypertension.

Authors:  Cristhiaan D Ochoa; Lunyin Yu; Essam Al-Ansari; Charles A Hales; Deborah A Quinn
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5.  Thrombospondin-1-induced smooth muscle cell chemotaxis and proliferation are dependent on transforming growth factor-β2 and hyaluronic acid synthase.

Authors:  Jeffrey J Stein; Chinenye Iwuchukwu; Kristopher G Maier; Vivian Gahtan
Journal:  Mol Cell Biochem       Date:  2013-10-09       Impact factor: 3.396

6.  In vitro and in vivo properties of distinct populations of amniotic fluid mesenchymal progenitor cells.

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Review 7.  Cardiovascular Effects Mediated by HMMR and CD44.

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  7 in total

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