Literature DB >> 19883181

Endogenous protection against oxidative stress caused by cisplatin: role of superoxide dismutase.

José Angel González-García1, Julián Nevado, José Ramón García-Berrocal, Carolina Sánchez-Rodríguez, Almudena Trinidad, Ricardo Sanz, Rafael Ramírez-Camacho.   

Abstract

CONCLUSION: The administration of cisplatin induces the activation of superoxide dismutase (SOD) as a response to oxidative stress in the cochleae of Sprague-Dawley rats and this activation is proportional to the activation of the intrinsic pathway of apoptosis.
OBJECTIVES: To determine the role of the antioxidant endogenous mechanism in the preservation of cochlear integrity and function in an experimental model of cisplatin ototoxicity.
METHODS: Sixteen Sprague-Dawley rats were studied at 7 days after intraperitoneal injection of CDDP (n = 8) or 10 ml/kg NaCl 0.9% w/v in the control group (n = 8) by means of auditory steady-state responses. These findings were compared with the expression of SOD and caspase-3/7 and caspase-9 activities.
RESULTS: Groups receiving cisplatin showed increased auditory thresholds after injection of cisplatin and control groups maintained normal hearing. Measurements of caspase-3/7 and caspase-9 showed a significant increase in cisplatin-treated rats. A significantly increased activity of total SOD in whole cochlear extracts was observed in animals from the CDDP groups vs control animals. Likewise, differences between CDDP groups were also statistically significant.

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Year:  2010        PMID: 19883181     DOI: 10.3109/00016480903311203

Source DB:  PubMed          Journal:  Acta Otolaryngol        ISSN: 0001-6489            Impact factor:   1.494


  7 in total

Review 1.  Effect of intratympanic dimethyl sulphoxide (DMSO) in an in vivo model of cisplatin-related ototoxicity.

Authors:  A Roldán-Fidalgo; A Trinidad; A Rodríguez-Valiente; J R García-Berrocal; I Millán; M J Coronado; R Ramírez-Camacho
Journal:  Eur Arch Otorhinolaryngol       Date:  2014-03-08       Impact factor: 2.503

2.  Some Ototoxic Drugs Destroy Cochlear Support Cells Before Damaging Sensory Hair Cells.

Authors:  Dalian Ding; Jianhui Zhang; Haiyan Jiang; Weijun Xuan; Weidong Qi; Richard Salvi
Journal:  Neurotox Res       Date:  2020-01-29       Impact factor: 3.911

3.  Blocking caspase-3-dependent pathway preserves hair cells from salicylate-induced apoptosis in the guinea pig cochlea.

Authors:  Hao Feng; Shi-Hua Yin; An-Zhou Tang
Journal:  Mol Cell Biochem       Date:  2011-04-19       Impact factor: 3.396

Review 4.  Reactive oxygen species in apoptosis induced by cisplatin: review of physiopathological mechanisms in animal models.

Authors:  Celia Casares; Rafael Ramírez-Camacho; Almudena Trinidad; Amaya Roldán; Eduardo Jorge; José Ramón García-Berrocal
Journal:  Eur Arch Otorhinolaryngol       Date:  2012-05-15       Impact factor: 2.503

Review 5.  Current Strategies to Combat Cisplatin-Induced Ototoxicity.

Authors:  Dehong Yu; Jiayi Gu; Yuming Chen; Wen Kang; Xueling Wang; Hao Wu
Journal:  Front Pharmacol       Date:  2020-07-03       Impact factor: 5.810

6.  Ursolic acid protects against cisplatin‑induced ototoxicity by inhibiting oxidative stress and TRPV1‑mediated Ca2+‑signaling.

Authors:  Yang Di; Tao Xu; Yuan Tian; Tingting Ma; Donghao Qu; Yan Wang; Yuhan Lin; Dongyan Bao; Li Yu; Shuangyue Liu; Aimei Wang
Journal:  Int J Mol Med       Date:  2020-06-04       Impact factor: 4.101

Review 7.  The Use of Nanoparticles in Otoprotection.

Authors:  Maurizio Barbara; Valerio Margani; Edoardo Covelli; Chiara Filippi; Luigi Volpini; Ola M El-Borady; Maged El-Kemary; Saad Elzayat; Haitham H Elfarargy
Journal:  Front Neurol       Date:  2022-06-29       Impact factor: 4.086

  7 in total

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