Literature DB >> 19880237

Growth inhibition of human ovarian cancer cells by blocking STAT3 activation with small interfering RNA.

Liying Cai1, Guangmei Zhang, Xiaojing Tong, Qi You, Yuan An, Yuguang Wang, Lei Guo, Tianzhen Wang, Daling Zhu, Jianhua Zheng.   

Abstract

OBJECTIVE: Signal transducer and activator of transcription 3 (STAT3) are constitutively activated in a variety of cancers and it is a common feature of ovarian cancer. Thus, STAT3 represents a promising molecular target for tumor therapy. We applied a DNA vector-based STAT3-specific RNA interference approach which specifically blocks over-activated STAT3, to treat human ovarian cancer cells, and evaluated the cellular proliferation ability and investigated the molecular mechanisms in vitro. STUDY
DESIGN: A DNA vector-based RNA interference approach was used to knockdown STAT3 expression in human ovarian cancer cells in vitro.
RESULTS: The STAT3 siRNA down-regulated the expression of cyclin D1, survivin, and VEGF in ovarian cancer cells both at transcription and translation levels. Inhibition of STAT3 and its related genes was accompanied by growth suppression and induction of apoptosis in cancer cells in vitro.
CONCLUSIONS: These data indicate that STAT3 signaling is a promising molecular target for ovarian cancer therapy.

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Year:  2010        PMID: 19880237     DOI: 10.1016/j.ejogrb.2009.09.018

Source DB:  PubMed          Journal:  Eur J Obstet Gynecol Reprod Biol        ISSN: 0301-2115            Impact factor:   2.435


  16 in total

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