Literature DB >> 19833354

Which is better to preserve pulmonary function: short-term or prolonged leukocyte depletion during cardiopulmonary bypass?

Kaiyu Tao1, Qi An, Ke Lin, Raphael C Lui, Xiaodong Wu, Jing Zhou, Lei Du.   

Abstract

OBJECTIVES: Neutrophils are crucial in the development of acute lung injuries during cardiopulmonary bypass. However, the efficacy of leukocyte depletion on pulmonary protection remains controversial, possibly owing to different filtration strategies used in the literature. In this study, we investigated whether short-term leukocyte depletion strategy is more efficacious than prolonged leukocyte depletion in preserving pulmonary function.
METHODS: Eighteen adult dogs were randomized equally into 3 groups. Leukocyte-depleting filters were used for 10 minutes in the LD-S group, throughout cardiopulmonary bypass in the LD-T group, and not used in the control group. Neutrophil counts, elastase, and interleukin-8 concentrations in plasma, myeloperoxidase and interleukin-8 concentrations in pulmonary tissue, and pulmonary vascular resistance and oxygen index were determined to evaluate the inflammatory response and damage to pulmonary function.
RESULTS: Although the neutrophil count and pulmonary parenchymal myeloperoxidase contents were significantly lower in both LD-S and LD-T groups than that in the control group, lower pulmonary parenchymal interleukin-8 level, lower pulmonary vascular resistance (113 +/- 33 dyne x s/cm(5)), higher oxygen index (366 +/- 82.3 mm Hg), and thinner alveolus wall thickness were seen only in the LD-S group, and the pulmonary parenchymal interleukin-8 levels were also lower in the LD-S group after cardiopulmonary bypass. The plasma elastase and interleukin-8 levels were significantly lower in the LD-S group, but they were significantly higher in the LD-T group compared with the control group after cardiopulmonary bypass.
CONCLUSIONS: Short-term rather than prolonged leukocyte depletion during cardiopulmonary bypass appears to be more efficacious in protecting pulmonary function via attenuation of the extracorporeal circulation-induced inflammatory response.

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Year:  2009        PMID: 19833354     DOI: 10.1016/j.jtcvs.2009.07.059

Source DB:  PubMed          Journal:  J Thorac Cardiovasc Surg        ISSN: 0022-5223            Impact factor:   5.209


  5 in total

1.  Is there a role of TNFR1 in acute lung injury cases associated with extracorporeal circulation?

Authors:  Yu Zhao; Chong-wei Zhang; Wen-jing Zhou; Jiao Chen; Nan-fu Luo; Li-na Gong; Lei Du; Jing Zhou
Journal:  J Zhejiang Univ Sci B       Date:  2014-03       Impact factor: 3.066

2.  Novel Leukocyte Modulator Device Reduces the Inflammatory Response to Cardiopulmonary Bypass.

Authors:  Kimberly A Johnston; Angela J Westover; Alvaro Rojas-Pena; Jonathan W Haft; John M Toomasian; Thomas Johnson; Deborah A Buffington; H David Humes
Journal:  ASAIO J       Date:  2019 May/Jun       Impact factor: 2.872

3.  Long-term leukocyte filtration should be avoided during extracorporeal circulation.

Authors:  Jiali Tang; Kaiyu Tao; Jing Zhou; Chongwei Zhang; Lina Gong; Nanfu Luo; Lei Du
Journal:  Mediators Inflamm       Date:  2013-12-26       Impact factor: 4.711

4.  Leukocyte depletion during CPB: effects on inflammation and lung function.

Authors:  Célio Gomes de Amorim; Luiz Marcelo Sá Malbouisson; Francisco Costa da Silva; Alfredo Inácio Fiorelli; Caroline Kameio Fernandes Murakami; Maria José Carvalho Carmona
Journal:  Inflammation       Date:  2014-02       Impact factor: 4.092

Review 5.  Managing the inflammatory response after cardiopulmonary bypass: review of the studies in animal models.

Authors:  Gabriel Romero Liguori; Alexandre Fligelman Kanas; Luiz Felipe Pinho Moreira
Journal:  Rev Bras Cir Cardiovasc       Date:  2014 Jan-Mar
  5 in total

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