Literature DB >> 19830844

Transforming growth factor-beta(1) in bronchoalveolar lavage fluid from children with cystic fibrosis.

William T Harris1, Marianne S Muhlebach, Robert A Oster, Michael R Knowles, Terry L Noah.   

Abstract

RATIONALE: Transforming factor beta(1) (TGF-beta(1)) genetic polymorphisms have been identified as a modifier of cystic fibrosis (CF) lung disease severity. However, few data link TGF-beta(1) protein levels and clinical markers of CF lung disease severity.
OBJECTIVES: To determine the association between protein levels of TGF-beta(1) in pediatric CF bronchoalveolar lavage fluid (BALF) and clinical parameters of CF lung disease severity.
METHODS: Total TGF-beta(1) was measured in BALF from 30 pediatric CF patients and 12 non-CF disease controls undergoing clinically indicated flexible bronchoscopy, and compared to four indicators of clinical disease: infection, inflammation, pulmonary function, and recent/recurrent hospitalization.
RESULTS: TGF-beta(1) was elevated in CF BALF compared to non-CF controls (135 +/- 15 pg/ml vs. 57 +/- 10 pg/ml, P < 0.01). In CF BALF, increased TGF-beta(1) was associated with elevated BALF PMN % (r = 0.67, P < 0.01). BALF TGF-beta(1) was increased in CF subjects whose FEV(1) after the completion of antibiotic therapy remained below CF age-normative median values (205.9 +/- 20.5 pg/ml vs. 106.4 +/- 24.0, P = 0.01). BALF TGF-beta(1) was increased in CF children hospitalized in the previous year compared to those not recently hospitalized (169.9 +/- 21.6 pg/ml vs. 107.5 +/- 17.5 pg/ml, P = 0.04). Neither the presence of a bacterial pathogen nor bacterial quantity was associated with BALF TGF-beta(1).
CONCLUSIONS: In CF, BALF TGF-beta(1) is elevated compared to non-CF controls. Increased BALF TGF-beta(1) is associated with neutrophilic inflammation, diminished lung function and recent hospitalization. Further investigation is needed to address mechanisms behind these associations.

Entities:  

Mesh:

Substances:

Year:  2009        PMID: 19830844     DOI: 10.1002/ppul.21079

Source DB:  PubMed          Journal:  Pediatr Pulmonol        ISSN: 1099-0496


  26 in total

1.  Losartan Rescues Inflammation-related Mucociliary Dysfunction in Relevant Models of Cystic Fibrosis.

Authors:  Michael D Kim; Nathalie Baumlin; Makoto Yoshida; Deepika Polineni; Sebastian F Salathe; Joseph K David; Charles A Peloquin; Adam Wanner; John S Dennis; Juliette Sailland; Philip Whitney; Frank T Horrigan; Juan R Sabater; William M Abraham; Matthias Salathe
Journal:  Am J Respir Crit Care Med       Date:  2020-02-01       Impact factor: 21.405

2.  Subacute TGFβ Exposure Drives Airway Hyperresponsiveness in Cystic Fibrosis Mice through the PI3K Pathway.

Authors:  Elizabeth L Kramer; Satish K Madala; Kristin M Hudock; Cynthia Davidson; John P Clancy
Journal:  Am J Respir Cell Mol Biol       Date:  2020-05       Impact factor: 6.914

3.  TGF-β1 Suppresses the Type I IFN Response and Induces Mitochondrial Dysfunction in Alveolar Macrophages.

Authors:  Jocelyn R Grunwell; Samantha M Yeligar; Susan Stephenson; Xiao Du Ping; Theresa W Gauthier; Anne M Fitzpatrick; Lou Ann S Brown
Journal:  J Immunol       Date:  2018-02-02       Impact factor: 5.422

Review 4.  The role of microRNAs in chronic respiratory disease: recent insights.

Authors:  Lindsay R Stolzenburg; Ann Harris
Journal:  Biol Chem       Date:  2018-02-23       Impact factor: 3.915

Review 5.  TGFβ as a therapeutic target in cystic fibrosis.

Authors:  Elizabeth L Kramer; John P Clancy
Journal:  Expert Opin Ther Targets       Date:  2017-12-13       Impact factor: 6.902

6.  Association of Serum TGF-β1 Levels with Different Clinical Phenotypes of Cystic Fibrosis Exacerbation.

Authors:  Swati Sagwal; Anil Chauhan; Jyotdeep Kaur; Rajendra Prasad; Meenu Singh; Manvi Singh
Journal:  Lung       Date:  2020-01-09       Impact factor: 2.584

7.  Subacute TGFβ expression drives inflammation, goblet cell hyperplasia, and pulmonary function abnormalities in mice with effects dependent on CFTR function.

Authors:  Elizabeth L Kramer; William D Hardie; Satish K Madala; Cynthia Davidson; John P Clancy
Journal:  Am J Physiol Lung Cell Mol Physiol       Date:  2018-06-07       Impact factor: 5.464

8.  Airway Surface Dehydration by Transforming Growth Factor β (TGF-β) in Cystic Fibrosis Is Due to Decreased Function of a Voltage-dependent Potassium Channel and Can Be Rescued by the Drug Pirfenidone.

Authors:  Dahis Manzanares; Stefanie Krick; Nathalie Baumlin; John S Dennis; Jean Tyrrell; Robert Tarran; Matthias Salathe
Journal:  J Biol Chem       Date:  2015-09-03       Impact factor: 5.157

9.  Constrictive Bronchiolitis in Cystic Fibrosis Adolescents with Refractory Pulmonary Decline.

Authors:  William T Harris; J Todd Boyd; Gary L McPhail; Alan S Brody; Rhonda D Szczesniak; Leslie L Korbee; Michael L Baker; John P Clancy
Journal:  Ann Am Thorac Soc       Date:  2016-12

10.  miR-1343 attenuates pathways of fibrosis by targeting the TGF-β receptors.

Authors:  Lindsay R Stolzenburg; Sarah Wachtel; Hong Dang; Ann Harris
Journal:  Biochem J       Date:  2015-11-05       Impact factor: 3.857

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.