Literature DB >> 19816711

Inhibitory effect of cucurbitacin E on pancreatic cancer cells growth via STAT3 signaling.

Chunyan Sun1, Meixia Zhang, Xiaolei Shan, Xueying Zhou, Jiao Yang, Yanli Wang, Jesse Li-Ling, Yihui Deng.   

Abstract

PURPOSE: Pancreatic cancer has been a serious disease worldwide for its high mortality. Cucurbitacin E is a member of triterpenoid family isolated from plants showing antiproliferative activity on various cancer cells. In this study, we have explored whether cucurbitacin E also has an anti-tumor effect on pancreatic cancer cells.
METHODS: Human pancreatic cancer cells PANC-1 were used to explore the effect and possible mechanisms of cucurbitacin E on cell cycle progression, apoptosis and proliferation.
RESULTS: Cucurbitacin E has inhibited the growth of PANC-1 cells in a dose- and time-dependent manner, and has caused accumulation of cells at the G(2)/M phase as well as apoptosis. Western blotting also showed that cucurbitacin E treatment can inhibit STAT3 phosphorylation while upregulate p53 expression.
CONCLUSIONS: Our results suggested that cucurbitacin E may be an effective regimen for the chemotherapy of pancreatic cancer.

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Year:  2009        PMID: 19816711     DOI: 10.1007/s00432-009-0698-x

Source DB:  PubMed          Journal:  J Cancer Res Clin Oncol        ISSN: 0171-5216            Impact factor:   4.553


  35 in total

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4.  RNA interference-mediated signal transducers and activators of transcription 3 gene silencing inhibits invasion and metastasis of human pancreatic cancer cells.

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5.  Targeted constitutive activation of signal transducer and activator of transcription 3 in human hepatocellular carcinoma cells by cucurbitacin B.

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Review 7.  Targeting AMPK signaling in combating ovarian cancers: opportunities and challenges.

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Review 8.  Herbals and Plants in the Treatment of Pancreatic Cancer: A Systematic Review of Experimental and Clinical Studies.

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