Literature DB >> 19724199

Subconjunctival and intracorneal bevacizumab injection for corneal neovascularization in lipid keratopathy.

Joo Youn Oh1, Mee Kum Kim, Won Ryang Wee.   

Abstract

PURPOSE: The purpose of this study was to evaluate the efficacy and safety of subconjunctival and corneal intrastromal bevacizumab injections on corneal neovascularization in patients with lipid keratopathy.
METHODS: Three eyes of 3 patients with lipid keratopathy accompanied by corneal neovascularization received at least 2 subconjunctival and corneal intrastromal injections of bevacizumab (1.25 mg/0.05 mL, respectively) at 1-month intervals. Ophthalmic examinations, including visual acuity, tonometry, slit lamp biomicroscopy, and corneal topography, were performed before injection and every month thereafter.
RESULTS: All patients had marked regression of corneal neovascularization. Reduction in lipid deposition was noted in one patient. There were no adverse ocular or systemic events, except for a small intracorneal hemorrhage noted in one patient, which quickly and spontaneously cleared.
CONCLUSION: Subconjunctival and intracorneal bevacizumab is a well-tolerated and effective method for obliterating corneal neovascularization in lipid keratopathy.

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Year:  2009        PMID: 19724199     DOI: 10.1097/ICO.0b013e31819839f9

Source DB:  PubMed          Journal:  Cornea        ISSN: 0277-3740            Impact factor:   2.651


  12 in total

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3.  Intrastromal delivery of bevacizumab using microneedles to treat corneal neovascularization.

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Review 6.  Ocular delivery of macromolecules.

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7.  Corneal endothelial safety following subconjunctival and intrastromal injection of bevacizumab for corneal neovascularization.

Authors:  Alejandro Lichtinger; Sonia N Yeung; Peter Kim; Maoz D Amiran; Uri Elbaz; Allan R Slomovic
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8.  Safety of bevacizumab on extraocular muscle in a rabbit model.

Authors:  Jae Ho Jung; Jung Hoon Lee; Ji Eun Lee; Hee Young Choi
Journal:  Korean J Ophthalmol       Date:  2012-07-24

9.  Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis.

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Journal:  Theor Biol Med Model       Date:  2012-04-10       Impact factor: 2.432

10.  Prophylactic effect of topical silica nanoparticles as a novel antineovascularization agent for inhibiting corneal neovascularization following chemical burn.

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Journal:  Adv Biomed Res       Date:  2015-06-04
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