Literature DB >> 19671315

[Hepatitis B virus X protein upregulates the transcription activity of zinc finger transcription factor SNAI1].

Qian Lu1, Jun Yan, Yan Xiong, Lei Cai, Xiao-wu Li, Jia-hong Dong.   

Abstract

OBJECTIVE: To investigate whether Hepatitis B virus X protein (HBx) regulates the transcription activity of zinc finger transcription factor SNAI1, and to define the main regulation domain.
METHODS: A series of 5' truncated promoters of SNAI1 were amplified and subcloned into the pGL3-Basic vector to construct promoter luciferase report plasmids. The promoter luciferase report plasmids were cotransfected with HBx recombinant adenovirus and pRL-TK plasmid into human hepatocellular cancer cells of the line SMMC7721. Luciferase activity was measured, and relative luciferase activity was calculated.
RESULTS: Luciferase reporter plasmids containing 5' flanking deletion of SNAI1 promoter were successfully constructed. HBx obviously upregulated the activity of the SNAI1 promoter (-869 - +59), other than the SNAI1 promoter (-514 - +59) and SNAI1 promoter (-194 - +59).
CONCLUSION: HBx can stimulate the transcription of SNAI1, and the transcription regulation domain is located in the segment from -869 - -514 of the SNAI1 promoter.

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Year:  2009        PMID: 19671315

Source DB:  PubMed          Journal:  Zhonghua Yi Xue Za Zhi        ISSN: 0376-2491


  1 in total

Review 1.  Relationship between epithelial-to-mesenchymal transition and the inflammatory microenvironment of hepatocellular carcinoma.

Authors:  Long Yan; Feng Xu; Chao-Liu Dai
Journal:  J Exp Clin Cancer Res       Date:  2018-08-29
  1 in total

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