Literature DB >> 1963586

Genomic integration as a novel mechanism of EBV persistence.

E A Hurley1, J A McNeil, J B Lawrence, D A Thorley-Lawson.   

Abstract

Mesh：

Year:  1990        PMID： 1963586     DOI： 10.1007/978-3-642-75889-8_45

Source DB:  PubMed          Journal:  Curr Top Microbiol Immunol        ISSN： 0070-217X            Impact factor:   4.291


× No keyword cloud information.
  4 in total

1.  Epstein-Barr virus (EBV)-negative B-lymphoma cell lines for clonal isolation and replication of EBV recombinants.

Authors:  A Marchini; R Longnecker; E Kieff
Journal:  J Virol       Date:  1992-08       Impact factor: 5.103

2.  Germinal center B cells latently infected with Epstein-Barr virus proliferate extensively but do not increase in number.

Authors:  Jill E Roughan; Charles Torgbor; David A Thorley-Lawson
Journal:  J Virol       Date:  2009-11-04       Impact factor: 5.103

3.  Small molecule inhibition of Epstein-Barr virus nuclear antigen-1 DNA binding activity interferes with replication and persistence of the viral genome.

Authors:  Eun Kyung Lee; Sun Young Kim; Ka-Won Noh; Eun Hye Joo; Bo Zhao; Elliott Kieff; Myung-Soo Kang
Journal:  Antiviral Res       Date:  2014-01-31       Impact factor: 5.970

4.  ERK2 phosphorylation of EBNA1 serine 383 residue is important for EBNA1-dependent transactivation.

Authors:  Ka-Won Noh; Jihyun Park; Eun Hye Joo; Eun Kyung Lee; Eun Young Choi; Myung-Soo Kang
Journal:  Oncotarget       Date:  2016-05-03
  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.