Literature DB >> 19624714

Frontal and temporal volumes in Childhood Absence Epilepsy.

Rochelle Caplan1, Jennifer Levitt, Prabha Siddarth, Keng Nei Wu, Suresh Gurbani, Raman Sankar, W Donald Shields.   

Abstract

PURPOSE: This study compared frontotemporal brain volumes in children with childhood absence epilepsy (CAE) to age- and gender-matched children without epilepsy. It also examined the association of these volumes with seizure, demographic, perinatal, intelligence quotient (IQ), and psychopathology variables.
METHODS: Twenty-six children with CAE, aged 7.5-11.8 years, and 37 children without epilepsy underwent brain magnetic resonance imaging (MRI) scans at 1.5 Tesla. Tissue was segmented, and total brain, frontal lobe, frontal parcellations, and temporal lobe volumes were computed. All children had IQ testing and structured psychiatric interviews. Parents provided seizure, perinatal, and behavioral information on each child.
RESULTS: The CAE group had significantly smaller gray matter volumes of the left orbital frontal gyrus as well as both left and right temporal lobes compared to the age- and gender-matched children without epilepsy. In the CAE group these volumes were related to age, gender, ethnicity, and pregnancy complications but not to seizure, IQ, and psychopathology variables. In the group of children without epilepsy, however, the volumes were related to IQ.
CONCLUSION: These findings suggest that CAE impacts brain development in regions implicated in behavior, cognition, and language. In addition to supporting the cortical focus theory of CAE, these findings also imply that CAE is not a benign disorder.

Entities:  

Mesh:

Year:  2009        PMID: 19624714     DOI: 10.1111/j.1528-1167.2009.02198.x

Source DB:  PubMed          Journal:  Epilepsia        ISSN: 0013-9580            Impact factor:   5.864


  29 in total

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5.  Regional brain volumes and cognition in childhood epilepsy: does size really matter?

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6.  Default mode network abnormalities in idiopathic generalized epilepsy.

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Review 8.  Quantifying the deficit-imaging neurobehavioural impairment in childhood epilepsy.

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10.  The neuropsychological and academic substrate of new/recent-onset epilepsies.

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