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Literature DB >> 1961223

Seminars in medicine of the Beth Israel Hospital, Boston. beta-Amyloid and the pathogenesis of Alzheimer's disease.

B A Yankner¹, M M Mesulam.

Abstract

Entities: Disease

Mesh：

Substances：
Amyloid beta-Peptides

Year: 1991 PMID： 1961223 DOI： 10.1056/NEJM199112263252605

Source DB: PubMed Journal: N Engl J Med ISSN： 0028-4793 Impact factor: 91.245

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Cited

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36 in total

1. Adenosine 3',5'-cyclic monophosphate increases processing of amyloid precursor protein (APP) to beta-amyloid in neuroblastoma cells without changing APP levels or expression of APP mRNA.

Authors: A Kumar; F G La Rosa; A R Hovland; W C Cole; J Edwards-Prasad; K N Prasad
Journal: Neurochem Res Date: 1999-10 Impact factor: 3.996

2. The novel calpain inhibitor A-705253 potently inhibits oligomeric beta-amyloid-induced dynamin 1 and tau cleavage in hippocampal neurons.

Authors: Roxana C Sinjoanu; Sara Kleinschmidt; Robert S Bitner; Jorge D Brioni; Achim Moeller; Adriana Ferreira
Journal: Neurochem Int Date: 2008-06-12 Impact factor: 3.921

3. Prostaglandins act as neurotoxin for differentiated neuroblastoma cells in culture and increase levels of ubiquitin and beta-amyloid.

Authors: K N Prasad; F G La Rosa; J E Prasad
Journal: In Vitro Cell Dev Biol Anim Date: 1998-03 Impact factor: 2.416

4. Alzheimer's-specific effects of soluble beta-amyloid on protein kinase C-alpha and -gamma degradation in human fibroblasts.

Authors: A Favit; M Grimaldi; T J Nelson; D L Alkon
Journal: Proc Natl Acad Sci U S A Date: 1998-05-12 Impact factor: 11.205

5. Caspase-3- and calpain-mediated tau cleavage are differentially prevented by estrogen and testosterone in beta-amyloid-treated hippocampal neurons.

Authors: S-Y Park; C Tournell; R C Sinjoanu; A Ferreira
Journal: Neuroscience Date: 2006-10-19 Impact factor: 3.590

Seminars in medicine of the Beth Israel Hospital, Boston. beta-Amyloid and the pathogenesis of Alzheimer's disease.

1. Adenosine 3',5'-cyclic monophosphate increases processing of amyloid precursor protein (APP) to beta-amyloid in neuroblastoma cells without changing APP levels or expression of APP mRNA.

2. The novel calpain inhibitor A-705253 potently inhibits oligomeric beta-amyloid-induced dynamin 1 and tau cleavage in hippocampal neurons.

3. Prostaglandins act as neurotoxin for differentiated neuroblastoma cells in culture and increase levels of ubiquitin and beta-amyloid.

4. Alzheimer's-specific effects of soluble beta-amyloid on protein kinase C-alpha and -gamma degradation in human fibroblasts.

5. Caspase-3- and calpain-mediated tau cleavage are differentially prevented by estrogen and testosterone in beta-amyloid-treated hippocampal neurons.

Review 6. Amyloid beta-peptide and oxidative cellular injury in Alzheimer's disease.

7. Risk of antipsychotic drug use in patients with Alzheimer's disease treated with rivastigmine.

8. Cell death in Alzheimer's disease evaluated by DNA fragmentation in situ.

9. Specific domains of beta-amyloid from Alzheimer plaque elicit neuron killing in human microglia.

10. Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.