Literature DB >> 19608203

Six cases of SCA3/MJD patients that mimic hereditary spastic paraplegia in clinic.

Yin-guang Wang1, Juan Du, Jun-ling Wang, Juan Chen, Chong Chen, Ying-ying Luo, Zhi-quan Xiao, Hong Jiang, Xin-xiang Yan, Kun Xia, Qian Pan, Bei-sha Tang, Lu Shen.   

Abstract

BACKGROUND: Spinocerebellar ataxia type 3/Machado-Joseph disease (SCA3/MJD) is an autosomal dominant neurodegenerative disease characterized by cerebellar ataxia associated with varying phenotypic variability. It was reported that a few of SCA3/MJD patients showed marked spastic paraplegia with or without cerebellar ataxia, which was partially first diagnosed as hereditary spastic paraplegia (HSP) and considered to be a new subtype (subtype V). But the data in China is still absent.
OBJECTIVE: To investigate the mutation frequency and clinical features of subtype V of SCA3/MJD in Chinese patients with HSP.
METHODS: Mutation detection of MJD1 gene was carried out in 46 AD-HSP families and 58 sporadic cases.
RESULTS: Expanded CAG repeats that ranged from 64 to 81 of MJD1 gene were found in six probands from 46 AD-HSP families (13%, 6/46). No abnormal repeat expansion was found in sporadic cases (0/58). The initial symptoms of six SCA3 cases were all spasticity in the lower limbs, and nystagmus, dysphagia and dysarthria that occurred with disease progression seemed more frequent than HSP.
CONCLUSION: Subtype V of SCA3/MJD is not rare in China, but it is hard to distinguish between HSP and SCA3/MJD only by clinical manifestation and MRI, and MJD1 gene should be detected routinely in the patients diagnosed as HSP in clinic.

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Year:  2009        PMID: 19608203     DOI: 10.1016/j.jns.2009.06.027

Source DB:  PubMed          Journal:  J Neurol Sci        ISSN: 0022-510X            Impact factor:   3.181


  11 in total

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Authors:  Maria do Carmo Costa; Henry L Paulson
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Review 3.  Machado-Joseph Disease: from first descriptions to new perspectives.

Authors:  Conceição Bettencourt; Manuela Lima
Journal:  Orphanet J Rare Dis       Date:  2011-06-02       Impact factor: 4.123

4.  C-terminal proline deletions in KCNC3 cause delayed channel inactivation and an adult-onset progressive SCA13 with spasticity.

Authors:  Swati Khare; Kira Galeano; Yalan Zhang; Jerelyn A Nick; Harry S Nick; S H Subramony; Jacinda Sampson; Leonard K Kaczmarek; Michael F Waters
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Review 5.  Cellular and molecular pathways triggering neurodegeneration in the spinocerebellar ataxias.

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Review 6.  Conventional MRI findings in hereditary degenerative ataxias: a pictorial review.

Authors:  Sirio Cocozza; Giuseppe Pontillo; Giovanna De Michele; Martina Di Stasi; Elvira Guerriero; Teresa Perillo; Chiara Pane; Anna De Rosa; Lorenzo Ugga; Arturo Brunetti
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7.  Spinocerebellar Atrophy Type-3 with Chiari Malformation in a Young Man: A Case Report.

Authors:  Sepideh Paybast; Mohsen Koosha; Dina Motamedi; Arman Habibi
Journal:  Adv J Emerg Med       Date:  2019-08-18

8.  Diagnosis, investigation and management of hereditary spastic paraplegias in the era of next-generation sequencing.

Authors:  Anke Hensiek; Stephen Kirker; Evan Reid
Journal:  J Neurol       Date:  2014-12-06       Impact factor: 4.849

9.  Epidemiological, clinical, and molecular characterization of Cuban families with spinocerebellar ataxia type 3/Machado-Joseph disease.

Authors:  Yanetza González-Zaldívar; Yaimeé Vázquez-Mojena; José M Laffita-Mesa; Luis E Almaguer-Mederos; Roberto Rodríguez-Labrada; Gilberto Sánchez-Cruz; Raúl Aguilera-Rodríguez; Tania Cruz-Mariño; Nalia Canales-Ochoa; Patrick MacLeod; Luis Velázquez-Pérez
Journal:  Cerebellum Ataxias       Date:  2015-02-21

10.  Striatal dopaminergic functioning in patients with sporadic and hereditary spastic paraplegias with parkinsonism.

Authors:  Ji Seon Kim; Jong Min Kim; Yu Kyeong Kim; Sang Eun Kim; Ji Young Yun; Beom S Jeon
Journal:  J Korean Med Sci       Date:  2013-10-31       Impact factor: 2.153

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