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Literature DB >> 19567746

Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.

Eduardo Ríos, Sandor Györke.

Abstract

Entities: Chemical

Mesh：

Substances：

Year: 2009 PMID： 19567746 PMCID： PMC2727014 DOI： 10.1113/jphysiol.2009.175083

Source DB: PubMed Journal: J Physiol ISSN： 0022-3751 Impact factor: 5.182

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17 in total

Review 1. Cardiac excitation-contraction coupling.

Authors: Donald M Bers
Journal: Nature Date: 2002-01-10 Impact factor: 49.962

Review 2. Local Ca(2+) signaling and EC coupling in heart: Ca(2+) sparks and the regulation of the [Ca(2+)](i) transient.

Authors: Silvia Guatimosim; Keith Dilly; L Fernando Santana; M Saleet Jafri; Eric A Sobie; W J Lederer
Journal: J Mol Cell Cardiol Date: 2002-08 Impact factor: 5.000

Review 3. Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles.

Authors: Susan Treves; Mirko Vukcevic; Marcin Maj; Raphael Thurnheer; Barbara Mosca; Francesco Zorzato
Journal: J Physiol Date: 2009-04-29 Impact factor: 5.182

Review 4. Deconstructing calsequestrin. Complex buffering in the calcium store of skeletal muscle.

Authors: Leandro Royer; Eduardo Ríos
Journal: J Physiol Date: 2009-04-29 Impact factor: 5.182

5. Silencing genes of sarcoplasmic reticulum proteins clarifies their roles in excitation-contraction coupling.

Authors: Gerhard Meissner; Ying Wang; Le Xu; Jerry P Eu
Journal: J Physiol Date: 2009-07-01 Impact factor: 5.182

Review 6. Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis.

Authors: Tracy J Pritchard; Evangelia G Kranias
Journal: J Physiol Date: 2009-04-29 Impact factor: 5.182

Review 7. Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle.

Authors: Robert T Dirksen
Journal: J Physiol Date: 2009-04-30 Impact factor: 5.182

Review 8. Triadin: what possible function 20 years later?

Authors: Isabelle Marty; Julien Fauré; Anne Fourest-Lieuvin; Stéphane Vassilopoulos; Sarah Oddoux; Julie Brocard
Journal: J Physiol Date: 2009-04-29 Impact factor: 5.182

Review 9. New roles of calsequestrin and triadin in cardiac muscle.

Authors: Björn C Knollmann
Journal: J Physiol Date: 2009-05-18 Impact factor: 5.182

Review 10. Calsequestrin-1: a new candidate gene for malignant hyperthermia and exertional/environmental heat stroke.

Authors: Feliciano Protasi; Cecilia Paolini; Marco Dainese
Journal: J Physiol Date: 2009-05-05 Impact factor: 5.182

6 in total

Review 1. Role of ryanodine receptor subtypes in initiation and formation of calcium sparks in arterial smooth muscle: comparison with striated muscle.

Authors: Kirill Essin; Maik Gollasch
Journal: J Biomed Biotechnol Date: 2009-12-08

Review 6. Improper Remodeling of Organelles Deputed to Ca²⁺ Handling and Aerobic ATP Production Underlies Muscle Dysfunction in Ageing.

Authors: Feliciano Protasi; Laura Pietrangelo; Simona Boncompagni
Journal: Int J Mol Sci Date: 2021-06-08 Impact factor: 5.923

6 in total

Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.

Review 1. Cardiac excitation-contraction coupling.

Review 2. Local Ca(2+) signaling and EC coupling in heart: Ca(2+) sparks and the regulation of the [Ca(2+)](i) transient.

Review 3. Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles.

Review 4. Deconstructing calsequestrin. Complex buffering in the calcium store of skeletal muscle.

5. Silencing genes of sarcoplasmic reticulum proteins clarifies their roles in excitation-contraction coupling.

Review 6. Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis.

Review 7. Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle.

Review 8. Triadin: what possible function 20 years later?

Review 9. New roles of calsequestrin and triadin in cardiac muscle.

Review 10. Calsequestrin-1: a new candidate gene for malignant hyperthermia and exertional/environmental heat stroke.

Review 1. Role of ryanodine receptor subtypes in initiation and formation of calcium sparks in arterial smooth muscle: comparison with striated muscle.

Review 2. The relationship between form and function throughout the history of excitation-contraction coupling.

Review 3. Cardiac Arrhythmias as Manifestations of Nanopathies: An Emerging View.

4. Characterization of a novel zebrafish model of SPEG-related centronuclear myopathy.

Review 5. Congenital myopathies: disorders of excitation-contraction coupling and muscle contraction.

Review 6. Improper Remodeling of Organelles Deputed to Ca²⁺ Handling and Aerobic ATP Production Underlies Muscle Dysfunction in Ageing.

Calsequestrin, triadin and more: the molecules that modulate calcium release in cardiac and skeletal muscle.

Review 1. Cardiac excitation-contraction coupling.

Review 2. Local Ca(2+) signaling and EC coupling in heart: Ca(2+) sparks and the regulation of the [Ca(2+)](i) transient.

Review 3. Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles.

Review 4. Deconstructing calsequestrin. Complex buffering in the calcium store of skeletal muscle.

5. Silencing genes of sarcoplasmic reticulum proteins clarifies their roles in excitation-contraction coupling.

Review 6. Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis.

Review 7. Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle.

Review 8. Triadin: what possible function 20 years later?

Review 9. New roles of calsequestrin and triadin in cardiac muscle.

Review 10. Calsequestrin-1: a new candidate gene for malignant hyperthermia and exertional/environmental heat stroke.

Review 1. Role of ryanodine receptor subtypes in initiation and formation of calcium sparks in arterial smooth muscle: comparison with striated muscle.

Review 2. The relationship between form and function throughout the history of excitation-contraction coupling.

Review 3. Cardiac Arrhythmias as Manifestations of Nanopathies: An Emerging View.

4. Characterization of a novel zebrafish model of SPEG-related centronuclear myopathy.

Review 5. Congenital myopathies: disorders of excitation-contraction coupling and muscle contraction.

Review 6. Improper Remodeling of Organelles Deputed to Ca2+ Handling and Aerobic ATP Production Underlies Muscle Dysfunction in Ageing.

Review 6. Improper Remodeling of Organelles Deputed to Ca²⁺ Handling and Aerobic ATP Production Underlies Muscle Dysfunction in Ageing.