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16 in total

1. Apigenin inhibits the TNFα-induced expression of eNOS and MMP-9 via modulating Akt signalling through oestrogen receptor engagement.

Authors: Daniela Palmieri; Patrizia Perego; Domenico Palombo
Journal: Mol Cell Biochem Date: 2012-08-17 Impact factor: 3.396

Review 2. The effects of oestrogens and their receptors on cardiometabolic health.

Authors: Eugenia Morselli; Roberta S Santos; Alfredo Criollo; Michael D Nelson; Biff F Palmer; Deborah J Clegg
Journal: Nat Rev Endocrinol Date: 2017-03-17 Impact factor: 43.330

Review 3. Sex differences in vascular physiology and pathophysiology: estrogen and androgen signaling in health and disease.

Authors: Austin C Boese; Seong C Kim; Ke-Jie Yin; Jean-Pyo Lee; Milton H Hamblin
Journal: Am J Physiol Heart Circ Physiol Date: 2017-06-16 Impact factor: 4.733

Review 4. The G protein-coupled estrogen receptor GPER/GPR30 as a regulator of cardiovascular function.

Authors: Matthias R Meyer; Eric R Prossnitz; Matthias Barton
Journal: Vascul Pharmacol Date: 2011-07-05 Impact factor: 5.773

Review 5. Back to your heart: ubiquitin proteasome system-regulated signal transduction.

Authors: Andrea L Portbury; Sarah M Ronnebaum; Makhosazane Zungu; Cam Patterson; Monte S Willis
Journal: J Mol Cell Cardiol Date: 2011-11-06 Impact factor: 5.000

Review 6. The G-protein-coupled estrogen receptor GPER in health and disease.

Authors: Eric R Prossnitz; Matthias Barton
Journal: Nat Rev Endocrinol Date: 2011-08-16 Impact factor: 43.330

7. 17β-Estradiol nongenomically induces vascular endothelial H₂S release by promoting phosphorylation of cystathionine γ-lyase.

Authors: Xingyan Xu; Qing Yan; Xiaoyun Liu; Ping Li; Xiaosa Li; Yiwen Chen; Tommaso Simoncini; Junxiu Liu; Dongxing Zhu; Xiaodong Fu
Journal: J Biol Chem Date: 2019-08-22 Impact factor: 5.157

ERalpha, ERbeta, and gpER: novel aspects of oestrogen receptor signalling in atherosclerosis.

1. Apigenin inhibits the TNFα-induced expression of eNOS and MMP-9 via modulating Akt signalling through oestrogen receptor engagement.

Review 2. The effects of oestrogens and their receptors on cardiometabolic health.

Review 3. Sex differences in vascular physiology and pathophysiology: estrogen and androgen signaling in health and disease.

Review 4. The G protein-coupled estrogen receptor GPER/GPR30 as a regulator of cardiovascular function.

Review 5. Back to your heart: ubiquitin proteasome system-regulated signal transduction.

Review 6. The G-protein-coupled estrogen receptor GPER in health and disease.

7. 17β-Estradiol nongenomically induces vascular endothelial H₂S release by promoting phosphorylation of cystathionine γ-lyase.

Review 8. Emerging roles of GPER in diabetes and atherosclerosis.

Review 9. Alike but not the same: anatomic heterogeneity of estrogen receptor-mediated vasodilation.

Review 10. Estrogen biology: new insights into GPER function and clinical opportunities.

ERalpha, ERbeta, and gpER: novel aspects of oestrogen receptor signalling in atherosclerosis.

1. Apigenin inhibits the TNFα-induced expression of eNOS and MMP-9 via modulating Akt signalling through oestrogen receptor engagement.

Review 2. The effects of oestrogens and their receptors on cardiometabolic health.

Review 3. Sex differences in vascular physiology and pathophysiology: estrogen and androgen signaling in health and disease.

Review 4. The G protein-coupled estrogen receptor GPER/GPR30 as a regulator of cardiovascular function.

Review 5. Back to your heart: ubiquitin proteasome system-regulated signal transduction.

Review 6. The G-protein-coupled estrogen receptor GPER in health and disease.

7. 17β-Estradiol nongenomically induces vascular endothelial H2S release by promoting phosphorylation of cystathionine γ-lyase.

Review 8. Emerging roles of GPER in diabetes and atherosclerosis.

Review 9. Alike but not the same: anatomic heterogeneity of estrogen receptor-mediated vasodilation.

Review 10. Estrogen biology: new insights into GPER function and clinical opportunities.

7. 17β-Estradiol nongenomically induces vascular endothelial H₂S release by promoting phosphorylation of cystathionine γ-lyase.