| Literature DB >> 19490402 |
William O Tatum1, Daniel D Bui, Edmund G Grant, Ryan Murtagh.
Abstract
Guillain-Barre syndrome (GBS) is the rubric encompassing highly variable phenotypic subgroups of acute, postinfectious, immune-mediated peripheral neuropathy. The hallmark of GBS phenomenology is a rapidly progressive ascending lower extremity weakness. GBS taxonomy includes a motor and sensory axonal neuropathy (AMSAN). Nitrous oxide (NO) abuse may create a pattern of neurological dysfunction almost identical to subacute combined degeneration. We report an adult with myeloneuropathy due to NO abuse that mimicked the presenting features of the GBS-subtype AMSAN.Entities:
Mesh:
Year: 2010 PMID: 19490402 DOI: 10.1111/j.1552-6569.2009.00388.x
Source DB: PubMed Journal: J Neuroimaging ISSN: 1051-2284 Impact factor: 2.486