Literature DB >> 19479851

Impaired differentiation and cytotoxicity of natural killer cells in systemic lupus erythematosus.

Yong-Wook Park1, Seung-Jung Kee, Young-Nan Cho, Eun-Hee Lee, Hwa-Youn Lee, Eun-Mi Kim, Min-Ho Shin, Jeong-Jin Park, Tae-Jong Kim, Shin-Seok Lee, Dae-Hyun Yoo, Hyung-Sik Kang.   

Abstract

OBJECTIVE: To determine the cytotoxicity of natural killer (NK) cells and the level of differentiation of hematopoietic stem cells (HSCs) into NK cells in systemic lupus erythematosus (SLE).
METHODS: Patients with SLE (n=108), rheumatoid arthritis (RA; n=90), Behçet's disease (n=39), or ankylosing spondylitis (n=41) and healthy control subjects (n=173) were enrolled in the study. NK cell levels, NK cell cytotoxicities, and lymphokine-activated killer (LAK) activities against K562 cells were measured by flow cytometry. Gene expression was assessed by reverse transcription-polymerase chain reaction. NK cells were differentiated from peripheral blood and bone marrow HSCs in vitro.
RESULTS: Percentages and absolute numbers of NK cells, cytotoxicities, and LAK activities were significantly lower in the peripheral blood of SLE and RA patients than in that of healthy controls. In particular, this NK cell deficiency was more prominent in patients with lupus nephritis and those with thrombocytopenia. Notably, purified NK cells derived from SLE patients, but not RA patients, were found to have lower cytotoxicities and LAK activities than those from healthy controls. This defect of NK cells in SLE patients was found to be related to lower numbers of NK precursors and to the down-regulation of perforin and granzyme in NK cells. The proliferative capacity of HSCs, the percentages of NK cells differentiated from HSCs, and NK cell cytotoxicities were significantly lower in SLE patients.
CONCLUSION: In SLE patients, circulating levels of NK cells were diminished and their cytotoxicities were impaired. Furthermore, the differentiation of HSCs into NK cells was found to be defective. These abnormalities possibly contribute to immune system dysregulation in SLE.

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Year:  2009        PMID: 19479851     DOI: 10.1002/art.24556

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  61 in total

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Review 4.  Could Lymphocyte Profiling be Useful to Diagnose Systemic Autoimmune Diseases?

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5.  NKG2D ligand overexpression in lupus nephritis correlates with increased NK cell activity and differentiation in kidneys but not in the periphery.

Authors:  Roberto Spada; José M Rojas; Sonia Pérez-Yagüe; Vladimir Mulens; Pablo Cannata-Ortiz; Rafael Bragado; Domingo F Barber
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6.  Association of killer cell immunoglobulin-like receptor and human leucocyte antigen-Cw gene combinations with systemic lupus erythematosus.

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Journal:  Clin Exp Immunol       Date:  2015-05       Impact factor: 4.330

7.  A subgroup of lupus patients with nephritis, innate T cell activation and low vitamin D is identified by the enhancement of circulating MHC class I-related chain A.

Authors:  M Pérez-Ferro; F I Romero-Bueno; C Serrano Del Castillo; I Mahillo; A Alvear; R Largo; G Herrero-Beaumont; O Sánchez-Pernaute
Journal:  Clin Exp Immunol       Date:  2019-02-27       Impact factor: 4.330

8.  Assay of T- and NK-cell subsets and the expression of NKG2A and NKG2D in patients with new-onset systemic lupus erythematosus.

Authors:  Wen-Xian Li; Hai-Feng Pan; Jian-Li Hu; Chang-Zhong Wang; Ning Zhang; Jing Li; Xiang-Pei Li; Jian-Hua Xu; Dong-Qing Ye
Journal:  Clin Rheumatol       Date:  2009-12-10       Impact factor: 2.980

9.  Differential microRNA profile and post-transcriptional regulation exist in systemic lupus erythematosus patients with distinct autoantibody specificities.

Authors:  Sudhir Kumar Chauhan; Vikas Vikram Singh; Richa Rai; Madhukar Rai; Geeta Rai
Journal:  J Clin Immunol       Date:  2014-05       Impact factor: 8.317

10.  Analysis of the CD161-expressing cell quantities and CD161 expression levels in peripheral blood natural killer and T cells of systemic lupus erythematosus patients.

Authors:  Yi-Lung Lin; Shih-Chang Lin
Journal:  Clin Exp Med       Date:  2015-11-21       Impact factor: 3.984

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