Arthur L Burnett1. 1. Johns Hopkins Medical Institutions, The James Buchanan Brady Urological Institute, Department of Urology, Baltimore, MD, USA. Electronic address: aburnett@jhmi.edu.
Abstract
INTRODUCTION: A scientific explanation remains elusive for many presentations of recurrent ischemic priapism. AIM: The aim of this article was to evaluate the possible clinical association between idiopathic priapism and anxiety disorders. METHODS: Twenty-one patients without identifiable, presumed etiologies for their priapism disorders presented consecutively to a single practitioner's clinical practice and underwent clinical history evaluation and management over a 2(1/2)-year interval. MAIN OUTCOME MEASURES: The main outcome is the documentation of patient demographics and clinical profiles relating to medical history and priapism presentations. RESULTS: Anxiety disorders including generalized anxiety, attention-deficit hyperactivity disorder, and obsessive-compulsive disorder were self-reported in 10 patients. CONCLUSIONS: The possible association between idiopathic priapism and anxiety disorders lends support for a central neurobiologic pathophysiology for the erectile disorder. Further neuropsychiatric studies of this clinical population is required to confirm the hypothesis.
INTRODUCTION: A scientific explanation remains elusive for many presentations of recurrent ischemic priapism. AIM: The aim of this article was to evaluate the possible clinical association between idiopathic priapism and anxiety disorders. METHODS: Twenty-one patients without identifiable, presumed etiologies for their priapism disorders presented consecutively to a single practitioner's clinical practice and underwent clinical history evaluation and management over a 2(1/2)-year interval. MAIN OUTCOME MEASURES: The main outcome is the documentation of patient demographics and clinical profiles relating to medical history and priapism presentations. RESULTS:Anxiety disorders including generalized anxiety, attention-deficit hyperactivity disorder, and obsessive-compulsive disorder were self-reported in 10 patients. CONCLUSIONS: The possible association between idiopathic priapism and anxiety disorders lends support for a central neurobiologic pathophysiology for the erectile disorder. Further neuropsychiatric studies of this clinical population is required to confirm the hypothesis.