Literature DB >> 19471255

Novel association of the interleukin 2-interleukin 21 region with inflammatory bowel disease.

Ana Márquez1, Gisela Orozco, Alfonso Martínez, Rogelio Palomino-Morales, Miguel Fernández-Arquero, Juan Luis Mendoza, Carlos Taxonera, Manuel Díaz-Rubio, María Gómez-García, Antonio Nieto, Miguel A López-Nevot, Emilio G de la Concha, Javier Martín, Elena Urcelay.   

Abstract

OBJECTIVES: Genome-wide association studies have reported the role of the interleukin (IL) 2-IL21 chromosomal region at 4q27 in several autoimmune conditions. Mice deficient in IL-2 develop a disease with clinical and histological similarity to ulcerative colitis (UC) in humans. Modest evidence of linkage with UC was tentatively proposed for the IL2 gene more than a decade ago. Therefore, we decide to investigate the association of polymorphisms in the IL-2 axis (IL2, IL2RA, and IL2RB genes) with inflammatory bowel diseases (IBDs).
METHODS: Seven hundred and twenty-eight white Spanish unrelated IBD patients (356 Crohn's disease (CD) and 372 UC) and 549 ethnically matched controls were included in a case-control study. In addition, a Spanish replication cohort with 562 CD and 430 UC patients and 1,310 controls were analyzed. Eight single-nucleotide polymorphisms previously associated with different autoimmune diseases were analyzed using TaqMan chemistry.
RESULTS: The IL2-rs6822844 polymorphism modified CD predisposition (P=0.002; odds ratio, OR (95% confidence interval, CI)=0.61 (0.44-0.84)); this was replicated in the other Spanish cohort, resulting in a strong protective effect of the minor allele in the merged samples (P=0.0002; OR (95% CI)=0.70 (0.58-0.85)). A similar effect of rs6822844 was detected for UC. Another marker, rs11938795, also showed evidence of an association with CD (P=0.006; OR (95% CI)=0.73 (0.58-0.92)).
CONCLUSIONS: Polymorphisms within the IL2-IL21 linkage disequilibrium (LD) block show a novel association with IBD, this is concordant with suggestive previous results of whole genome analyses in CD and type 1 diabetes. Our data agree with the effect previously observed for other conditions and delineate a shared underlying mechanism.

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Year:  2009        PMID: 19471255     DOI: 10.1038/ajg.2009.224

Source DB:  PubMed          Journal:  Am J Gastroenterol        ISSN: 0002-9270            Impact factor:   10.864


  17 in total

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9.  Only one independent genetic association with rheumatoid arthritis within the KIAA1109-TENR-IL2-IL21 locus in Caucasian sample sets: confirmation of association of rs6822844 with rheumatoid arthritis at a genome-wide level of significance.

Authors:  Jade E Hollis-Moffatt; Michael Chen-Xu; Ruth Topless; Nicola Dalbeth; Peter J Gow; Andrew A Harrison; John Highton; Peter B B Jones; Michael Nissen; Malcolm D Smith; Andre van Rij; Gregory T Jones; Lisa K Stamp; Tony R Merriman
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10.  Autoimmune diseases association study with the KIAA1109-IL2-IL21 region in a Tunisian population.

Authors:  Dorra Bouzid; Hajer Fourati; Ali Amouri; Isabel Marques; Olfa Abida; Nabil Tahri; Carlos Penha-Gonçalves; Hatem Masmoudi
Journal:  Mol Biol Rep       Date:  2014-07-19       Impact factor: 2.316

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