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Literature DB >> 19470039

Increased expression of CRTH2 on eosinophils in allergic skin diseases.

H Yahara, T Satoh, C Miyagishi, H Yokozeki.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 2009 PMID： 19470039 DOI： 10.1111/j.1468-3083.2009.03267.x

Source DB: PubMed Journal: J Eur Acad Dermatol Venereol ISSN： 0926-9959 Impact factor: 6.166

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Cited

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13 in total

1. Dual functions of prostaglandin D2 in murine contact hypersensitivity via DP and CRTH2.

Authors: Yoshihiro Yamamoto; Shinobu Otani; Hiroyuki Hirai; Kinya Nagata; Kosuke Aritake; Yoshihiro Urade; Shuh Narumiya; Hiroo Yokozeki; Masataka Nakamura; Takahiro Satoh
Journal: Am J Pathol Date: 2011-05-10 Impact factor: 4.307

2. A Comprehensive Approach to Urticaria: From Clinical Presentation to Modern Biological Treatments Through Pathogenesis.

Authors: Marco Folci; Giacomo Ramponi; Enrico Brunetta
Journal: Adv Exp Med Biol Date: 2021 Impact factor: 2.622

3. FcεRI, but not FcγR, signals induce prostaglandin D2 and E2 production from basophils.

Authors: Tsukasa Ugajin; Takahiro Satoh; Toru Kanamori; Kosuke Aritake; Yoshihiro Urade; Hiroo Yokozeki
Journal: Am J Pathol Date: 2011-06-25 Impact factor: 4.307

4. Th2 Cytokines and Atopic Dermatitis.

Authors: Eric B Brandt; Umasundari Sivaprasad
Journal: J Clin Cell Immunol Date: 2011-08-10

5. The prostaglandin D₂ receptor CRTH2 is important for allergic skin inflammation after epicutaneous antigen challenge.

Authors: Rui He; Michiko K Oyoshi; James Y T Wang; Martin R Hodge; Haoli Jin; Raif S Geha
Journal: J Allergy Clin Immunol Date: 2010-08-14 Impact factor: 10.793

6. Indomethacin inhibits eosinophil migration to prostaglandin D2 : therapeutic potential of CRTH2 desensitization for eosinophilic pustular folliculitis.

Authors: Naoko Kataoka; Takahiro Satoh; Aiko Hirai; Kazumi Saeki; Hiroo Yokozeki
Journal: Immunology Date: 2013-09 Impact factor: 7.397

Increased expression of CRTH2 on eosinophils in allergic skin diseases.

1. Dual functions of prostaglandin D2 in murine contact hypersensitivity via DP and CRTH2.

2. A Comprehensive Approach to Urticaria: From Clinical Presentation to Modern Biological Treatments Through Pathogenesis.

3. FcεRI, but not FcγR, signals induce prostaglandin D2 and E2 production from basophils.

4. Th2 Cytokines and Atopic Dermatitis.

5. The prostaglandin D₂ receptor CRTH2 is important for allergic skin inflammation after epicutaneous antigen challenge.

6. Indomethacin inhibits eosinophil migration to prostaglandin D2 : therapeutic potential of CRTH2 desensitization for eosinophilic pustular folliculitis.

Review 7. Current and emerging treatments for chronic spontaneous urticaria.

Review 8. Targeted Therapy for Chronıc Spontaneous Urtıcarıa: Ratıonale and Recent Progress.

9. New and Emerging Therapies for Pediatric Atopic Dermatitis.

Review 10. Looking forward to new targeted treatments for chronic spontaneous urticaria.