Literature DB >> 19466533

One hour reperfusion is enough to assess function and infarct size with TTC staining in Langendorff rat model.

R Ferrera1, S Benhabbouche, J C Bopassa, B Li, M Ovize.   

Abstract

BACKGROUND: There is not general agreement concerning the optimal time of reperfusion necessary to assess myocardial function and necrosis on isolated perfused heart model. Nevertheless, the study of cardioprotection (especially, pre- and postconditioning) requires a reliable and standardized assessment of myocardial necrosis.
OBJECTIVE: The objective of this study was thus to evaluate whether 1 h of reperfusion was sufficient to assess rat heart viability on Langendorff preparation. Isolated rat hearts (n = 30) underwent 40 min of global normothermic ischemia followed by 60 or 120 min Langendorff reperfusion. In each group, hearts were also randomly assigned into the 2 following sub-groups: postconditioning (PostC, consisting in 2 episodes of 30 s ischemia and 30 s reperfusion at the onset of reperfusion), and control (no intervention). Coronary flow, heart rate, dP/dt and rate-pressure-product were measured. Myocardial necrosis was assessed by TTC staining and LDH, CK release analysis.
RESULTS: Our results indicated that heart function tended to slightly decrease between 60 min and 120 min reperfusion. Infarct size was identical at 60 min and 120 min reperfusion, averaging 33-34% of total LV area in controls versus 17% in PostC (p < 0.001 between control and PostC groups). Similarly, the maximum of enzymatic releases (CK and LDH) measured in coronary effluents was at 60 min of reperfusion, followed by a progressive decrease at 90 min and 120 min. As expected, postconditioning limited enzymatic releases whatever the studied time of reperfusion.
CONCLUSION: In conclusion, we showed that prolonged reperfusion beyond 60 min was not useful for function assessment and did not change infarct size measurement, on Langendorff rat model of ischemia-reperfusion.

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Year:  2009        PMID: 19466533     DOI: 10.1007/s10557-009-6176-5

Source DB:  PubMed          Journal:  Cardiovasc Drugs Ther        ISSN: 0920-3206            Impact factor:   3.727


  33 in total

1.  Induction and Assessment of Ischemia-reperfusion Injury in Langendorff-perfused Rat Hearts.

Authors:  Daniel J Herr; Sverre E Aune; Donald R Menick
Journal:  J Vis Exp       Date:  2015-07-27       Impact factor: 1.355

Review 2.  Practical guidelines for rigor and reproducibility in preclinical and clinical studies on cardioprotection.

Authors:  Hans Erik Bøtker; Derek Hausenloy; Ioanna Andreadou; Salvatore Antonucci; Kerstin Boengler; Sean M Davidson; Soni Deshwal; Yvan Devaux; Fabio Di Lisa; Moises Di Sante; Panagiotis Efentakis; Saveria Femminò; David García-Dorado; Zoltán Giricz; Borja Ibanez; Efstathios Iliodromitis; Nina Kaludercic; Petra Kleinbongard; Markus Neuhäuser; Michel Ovize; Pasquale Pagliaro; Michael Rahbek-Schmidt; Marisol Ruiz-Meana; Klaus-Dieter Schlüter; Rainer Schulz; Andreas Skyschally; Catherine Wilder; Derek M Yellon; Peter Ferdinandy; Gerd Heusch
Journal:  Basic Res Cardiol       Date:  2018-08-17       Impact factor: 17.165

3.  Murine Left Anterior Descending (LAD) Coronary Artery Ligation: An Improved and Simplified Model for Myocardial Infarction.

Authors:  Karla Reichert; Bonnie Colantuono; Isabella McCormack; Fernanda Rodrigues; Vasile Pavlov; M Ruhul Abid
Journal:  J Vis Exp       Date:  2017-04-02       Impact factor: 1.355

4.  Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion.

Authors:  Sverre E Aune; Daniel J Herr; Santhosh K Mani; Donald R Menick
Journal:  J Mol Cell Cardiol       Date:  2014-03-13       Impact factor: 5.000

5.  Discrepancy in calcium release from the sarcoplasmic reticulum and intracellular acidic stores for the protection of the heart against ischemia/reperfusion injury.

Authors:  Aseel Khalaf; Fawzi Babiker
Journal:  J Physiol Biochem       Date:  2016-06-21       Impact factor: 4.158

6.  Cardiac-specific deletion of GCN5L1 restricts recovery from ischemia-reperfusion injury.

Authors:  Janet R Manning; Dharendra Thapa; Manling Zhang; Michael W Stoner; Javier Traba; Charles F McTiernan; Catherine Corey; Sruti Shiva; Michael N Sack; Iain Scott
Journal:  J Mol Cell Cardiol       Date:  2019-02-15       Impact factor: 5.000

7.  HDAC1 localizes to the mitochondria of cardiac myocytes and contributes to early cardiac reperfusion injury.

Authors:  Daniel J Herr; Mauhamad Baarine; Sverre E Aune; Xiaoyang Li; Lauren E Ball; John J Lemasters; Craig C Beeson; James C Chou; Donald R Menick
Journal:  J Mol Cell Cardiol       Date:  2017-12-07       Impact factor: 5.000

8.  Ischemic post-conditioning reduces infarct size of the in vivo rat heart: role of PI3-K, mTOR, GSK-3beta, and apoptosis.

Authors:  Claudia Wagner; Diana Tillack; Gregor Simonis; Ruth H Strasser; Christof Weinbrenner
Journal:  Mol Cell Biochem       Date:  2010-01-07       Impact factor: 3.396

9.  Dityrosine as a marker of acute myocardial infarction? Experiments with the isolated Langendorff heart.

Authors:  F Mayer; M Falk; R Huhn; F Behmenburg; S Ritz-Timme
Journal:  Int J Legal Med       Date:  2016-05-16       Impact factor: 2.686

10.  Oxygen surrounding the heart during ischemic conservation determines the myocardial injury during reperfusion.

Authors:  Yansheng Feng; Jean Chrisostome Bopassa
Journal:  Am J Cardiovasc Dis       Date:  2015-08-01
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