Literature DB >> 19465548

Extracellular SOD and aged blood vessels.

Tohru Fukai.   

Abstract

Mesh:

Substances:

Year:  2009        PMID: 19465548      PMCID: PMC5504446          DOI: 10.1152/ajpheart.00425.2009

Source DB:  PubMed          Journal:  Am J Physiol Heart Circ Physiol        ISSN: 0363-6135            Impact factor:   4.733


× No keyword cloud information.
  26 in total

1.  Aging: a theory based on free radical and radiation chemistry.

Authors:  D HARMAN
Journal:  J Gerontol       Date:  1956-07

2.  Sad heart from no SOD.

Authors:  Maria Carolina Gongora; David G Harrison
Journal:  Hypertension       Date:  2007-11-19       Impact factor: 10.190

Review 3.  Extracellular superoxide dismutase: a regulator of nitric oxide bioavailability.

Authors:  T D Oury; B J Day; J D Crapo
Journal:  Lab Invest       Date:  1996-11       Impact factor: 5.662

4.  Extracellular superoxide dismutase is a major antioxidant in human fibroblasts and slows telomere shortening.

Authors:  Violeta Serra; Thomas von Zglinicki; Mario Lorenz; Gabriele Saretzki
Journal:  J Biol Chem       Date:  2002-12-09       Impact factor: 5.157

5.  Role of extracellular superoxide dismutase in hypertension.

Authors:  Maria Carolina Gongora; Zhenyu Qin; Karine Laude; Ha Won Kim; Louise McCann; J Rodney Folz; Sergey Dikalov; Tohru Fukai; David G Harrison
Journal:  Hypertension       Date:  2006-07-24       Impact factor: 10.190

Review 6.  Nitric oxide and peroxynitrite in health and disease.

Authors:  Pál Pacher; Joseph S Beckman; Lucas Liaudet
Journal:  Physiol Rev       Date:  2007-01       Impact factor: 37.312

Review 7.  Extracellular superoxide dismutase and cardiovascular disease.

Authors:  Tohru Fukai; Rodney J Folz; Ulf Landmesser; David G Harrison
Journal:  Cardiovasc Res       Date:  2002-08-01       Impact factor: 10.787

8.  Extracellular superoxide dismutase is a major determinant of nitric oxide bioavailability: in vivo and ex vivo evidence from ecSOD-deficient mice.

Authors:  Oliver Jung; Stefan L Marklund; Helmut Geiger; Thierry Pedrazzini; Rudi Busse; Ralf P Brandes
Journal:  Circ Res       Date:  2003-08-21       Impact factor: 17.367

9.  Novel mechanism for regulation of extracellular SOD transcription and activity by copper: role of antioxidant-1.

Authors:  Shinichi Itoh; Kiyoshi Ozumi; Ha Won Kim; Osamu Nakagawa; Ronald D McKinney; Rodney J Folz; Igor N Zelko; Masuko Ushio-Fukai; Tohru Fukai
Journal:  Free Radic Biol Med       Date:  2008-10-21       Impact factor: 7.376

10.  Pharmacological induction of vascular extracellular superoxide dismutase expression in vivo.

Authors:  Marc Oppermann; Vera Balz; Volker Adams; Vu Thao-Vi Dao; Murat Bas; Tatsiana Suvorava; Georg Kojda
Journal:  J Cell Mol Med       Date:  2008-12-24       Impact factor: 5.310

View more
  4 in total

1.  The effects of aging on pulmonary oxidative damage, protein nitration, and extracellular superoxide dismutase down-regulation during systemic inflammation.

Authors:  Marlene E Starr; Junji Ueda; Shoji Yamamoto; B Mark Evers; Hiroshi Saito
Journal:  Free Radic Biol Med       Date:  2010-11-17       Impact factor: 7.376

2.  Transcriptional and phenotypic changes in aorta and aortic valve with aging and MnSOD deficiency in mice.

Authors:  Carolyn M Roos; Michael Hagler; Bin Zhang; Elise A Oehler; Arman Arghami; Jordan D Miller
Journal:  Am J Physiol Heart Circ Physiol       Date:  2013-08-30       Impact factor: 4.733

3.  Farnesoid X receptor antagonizes JNK signaling pathway in liver carcinogenesis by activating SOD3.

Authors:  Yan-Dong Wang; Wei-Dong Chen; Cunbao Li; Cong Guo; Yanyan Li; Hui Qi; Hailing Shen; Jing Kong; Xuecheng Long; Frank Yuan; Xichun Wang; Wendong Huang
Journal:  Mol Endocrinol       Date:  2014-12-12

Review 4.  Endothelial aging associated with oxidative stress can be modulated by a healthy mediterranean diet.

Authors:  Carmen Marín; Elena M Yubero-Serrano; José López-Miranda; Francisco Pérez-Jiménez
Journal:  Int J Mol Sci       Date:  2013-04-24       Impact factor: 5.923

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.