Literature DB >> 19458338

Regulation of interphotoreceptor retinoid-binding protein (IRBP)-specific Th1 and Th17 cells in anterior chamber-associated immune deviation (ACAID).

Yan Cui1, Hui Shao, Deming Sun, Henry J Kaplan.   

Abstract

PURPOSE: Intracameral (anterior chamber) injection of antigen inhibits the development of delayed-type hypersensitivity, a phenomenon known as anterior chamber-associated immune deviation (ACAID). The authors investigated the effect of intracameral injection of interphotoreceptor retinoid-binding protein (IRPB) peptides on the development of IFN-gamma(+) and IL-17(+) pathogenic T cells.
METHODS: A uveitogenic (IRBP1-20) or nonuveitogenic (IRBP161-180) peptide was injected into the anterior chamber (AC) of B6 mice. Seven days later, the mice were primed with a pathogenic dose of IRBP1-20 in adjuvant. Thirteen days later, the pathogenic activity of the T cells isolated from the spleens of treated and untreated mice were compared, and the numbers of Th1 and Th17 T cells were assessed by intracellular staining. Regulatory T-cell activity was assessed by antibody staining and functional assays. The authors also compared the effect of inhibition on EAU of ocular injection to various sites, including the AC, the vitreous cavity, and the subretinal space.
RESULTS: Intraocular injection of the uveitogenic peptide (IRBP1-20), but not the nonuveitogenic peptide (IRBP161-180), inhibited the generation of IFN-gamma(+) and IL-17(+) uveitogenic T cells and the development of experimental autoimmune uveitis (EAU). AC administration of IRBP1-20, but not IRBP161-180, significantly decreased the number of circulating gammadelta T cells after subsequent systemic immunization with IRBP1-20. Absence of the gammadelta T-cell population prohibited the development of ACAID.
CONCLUSIONS: Injection of a uveitogenic peptide into the AC inhibited the development of EAU by regulation of Th1 and Th17 IRBP-specific T cells. The circulating gammadelta T-cell population was reduced and was associated with decreased activation of IL-17(+) uveitogenic T cells.

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Year:  2009        PMID: 19458338      PMCID: PMC3275438          DOI: 10.1167/iovs.09-3389

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  19 in total

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2.  Gammadelta T cells promote anterior chamber-associated immune deviation and immune privilege through their production of IL-10.

Authors:  Hossam M Ashour; Jerry Y Niederkorn
Journal:  J Immunol       Date:  2006-12-15       Impact factor: 5.422

3.  Characterization of IL-17+ interphotoreceptor retinoid-binding protein-specific T cells in experimental autoimmune uveitis.

Authors:  Yong Peng; Gencheng Han; Hui Shao; Yali Wang; Henry J Kaplan; Deming Sun
Journal:  Invest Ophthalmol Vis Sci       Date:  2007-09       Impact factor: 4.799

4.  Activation of invariant NKT cells ameliorates experimental ocular autoimmunity by a mechanism involving innate IFN-gamma production and dampening of the adaptive Th1 and Th17 responses.

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5.  Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages.

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8.  Intraocular injection of class II-restricted peptide induces an unexpected population of CD8 regulatory cells.

Authors:  M M Kosiewicz; J W Streilein
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9.  Severe chronic experimental autoimmune uveitis (EAU) of the C57BL/6 mouse induced by adoptive transfer of IRBP1-20-specific T cells.

Authors:  Hui Shao; Tianjiang Liao; Yan Ke; Hongsheng Shi; Henry J Kaplan; Deming Sun
Journal:  Exp Eye Res       Date:  2005-08-25       Impact factor: 3.467

10.  Infiltration of the inflamed eye by NKT cells in a rat model of experimental autoimmune uveitis.

Authors:  Hui Shao; Luc Van Kaer; Sheher L Sun; Henry J Kaplan; Deming Sun
Journal:  J Autoimmun       Date:  2003-08       Impact factor: 7.094

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  3 in total

1.  Activated gammadelta T cells promote the activation of uveitogenic T cells and exacerbate EAU development.

Authors:  Hong Nian; Hui Shao; Rebecca L O'Brien; Willi K Born; Henry J Kaplan; Deming Sun
Journal:  Invest Ophthalmol Vis Sci       Date:  2011-07-29       Impact factor: 4.799

2.  An A2B Adenosine Receptor Agonist Promotes Th17 Autoimmune Responses in Experimental Autoimmune Uveitis (EAU) via Dendritic Cell Activation.

Authors:  Mingjiazi Chen; Dongchun Liang; Aijun Zuo; Hui Shao; Henry J Kaplan; Deming Sun
Journal:  PLoS One       Date:  2015-07-06       Impact factor: 3.240

3.  Anterior chamber associated immune deviation used as a neuroprotective strategy in rats with spinal cord injury.

Authors:  Beatriz Pineda-Rodriguez; Diana Toscano-Tejeida; Elisa García-Vences; Roxana Rodriguez-Barrera; Adrian Flores-Romero; Daniela Castellanos-Canales; Gabriel Gutierrez-Ospina; Laura Castillo-Carvajal; Esperanza Meléndez-Herrera; Antonio Ibarra
Journal:  PLoS One       Date:  2017-11-30       Impact factor: 3.240

  3 in total

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