Literature DB >> 19448189

A genomewide association study of response to lithium for prevention of recurrence in bipolar disorder.

Roy H Perlis1, Jordan W Smoller, Manuel A R Ferreira, Andrew McQuillin, Nick Bass, Jacob Lawrence, Gary S Sachs, Vishwajit Nimgaonkar, Edward M Scolnick, Hugh Gurling, Pamela Sklar, Shaun Purcell.   

Abstract

OBJECTIVE: Lithium remains a first-line treatment for bipolar disorder, but the mechanisms by which it prevents the recurrence of mood episodes are not known. The authors utilized data from a genomewide association study to examine associations between single nucleotide polymorphisms (SNPs) and the outcome of lithium treatment in two cohorts of patients with bipolar I disorder or bipolar II disorder.
METHOD: The hazard for mood episode recurrence was examined among 1,177 patients with bipolar I disorder or bipolar II disorder, including 458 individuals treated with lithium carbonate or citrate, who were participants in the Systematic Treatment Enhancement Program for Bipolar Disorder (STEP-BD) cohort. SNPs showing the greatest evidence of association in Cox regression models were then examined for association with positive lithium response among 359 bipolar I or II disorder patients treated with lithium carbonate or citrate in a second cohort from the University College London.
RESULTS: The strongest association in the STEP-BD cohort (minimum p=5.5 x 10(-7)) was identified for a region on chromosome 10p15 (rs10795189). Of the regions showing suggestive evidence (p<5 x 10(-4)) of association with lithium response, five were further associated with positive lithium response in the University College London cohort, including SNPs in a region on chromosome 4q32 spanning a gene coding for the glutamate/alpha-amino-3-hydroxy-5-methyl-4-isoxazolpropionate (AMPA) receptor GRIA2.
CONCLUSIONS: Multiple novel loci merit further examination for association with lithium response in bipolar disorder patients, including one region that spans the GRIA2 gene, for which expression has been shown to be regulated by lithium treatment.

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Year:  2009        PMID: 19448189      PMCID: PMC3908470          DOI: 10.1176/appi.ajp.2009.08111633

Source DB:  PubMed          Journal:  Am J Psychiatry        ISSN: 0002-953X            Impact factor:   18.112


  33 in total

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2.  Age at onset, childhood psychopathology, and 2-year outcome in psychotic bipolar disorder.

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Review 7.  Antidepressants for bipolar depression: a systematic review of randomized, controlled trials.

Authors:  Harm J Gijsman; John R Geddes; Jennifer M Rendell; Willem A Nolen; Guy M Goodwin
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Review 8.  Rationale, design, and methods of the systematic treatment enhancement program for bipolar disorder (STEP-BD).

Authors:  Gary S Sachs; Michael E Thase; Michael W Otto; Mark Bauer; David Miklowitz; Stephen R Wisniewski; Philip Lavori; Barry Lebowitz; Mathew Rudorfer; Ellen Frank; Andrew A Nierenberg; Maurizio Fava; Charles Bowden; Terence Ketter; Lauren Marangell; Joseph Calabrese; David Kupfer; Jerrold F Rosenbaum
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1.  The International Consortium on Lithium Genetics (ConLiGen): an initiative by the NIMH and IGSLI to study the genetic basis of response to lithium treatment.

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9.  Pharmacogenetics of Anti-Diabetes Drugs.

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10.  Lithium: a key to the genetics of bipolar disorder.

Authors:  Cristiana Cruceanu; Martin Alda; Gustavo Turecki
Journal:  Genome Med       Date:  2009-08-19       Impact factor: 11.117

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