Literature DB >> 19447921

Reversible airflow obstruction in lymphangioleiomyomatosis.

Angelo M Taveira-DaSilva1, Wendy K Steagall2, Antoinette Rabel2, Olanda Hathaway2, Sergio Harari3, Roberto Cassandro3, Mario Stylianou4, Joel Moss2.   

Abstract

BACKGROUND: We previously reported that approximately one-fourth of patients with lymphangioleiomyomatosis (LAM) may respond to therapy with bronchodilators. However, the validity of those observations has been questioned. The aims of the present study were to determine the prevalence of reversible airflow obstruction in patients with LAM and to identify associated clinical and physiologic parameters.
METHODS: First, the clinical and physiologic characteristics of 235 patients were analyzed to determine the frequency of the response to albuterol during a total of 2,307 visits. Second, we prospectively evaluated the response to albuterol (2.5 mg) and ipratropium (500 mug) in 130 patients, and correlated their responses with their clinical and physiologic characteristics.
RESULTS: In the retrospective study, 51% of the patients responded at least once to bronchodilators; of these, 12% responded >/= 50% of the time. A higher frequency of positive bronchodilator responses was associated with greater rates of decline in FEV(1) and diffusing capacity of the lung for carbon monoxide (Dlco). In the prospective study, 39 patients (30%) responded to bronchodilators, including 12 to ipratropium, 9 to albuterol, and 18 to both. The prevalence of asthma and smoking in the 39 responders was not different from that seen in the 91 nonresponders. Patients who responded to ipratropium, albuterol, or both had significantly (p < 0.02) lower FEV(1) and Dlco, and a greater rate of FEV(1) decline (p = 0.044) and Dlco decline (p = 0.039) than patients who did not respond to these bronchodilators. After adjusting for FEV(1)/FVC ratio, Dlco decline also was greater in responders than in nonresponders (p = 0.009).
CONCLUSIONS: Patients with LAM may have partially reversible airflow obstruction. A positive response to bronchodilators is associated with an accelerated rate of decline in pulmonary function.

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Year:  2009        PMID: 19447921      PMCID: PMC2818851          DOI: 10.1378/chest.09-0624

Source DB:  PubMed          Journal:  Chest        ISSN: 0012-3692            Impact factor:   9.410


  31 in total

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3.  Standardisation of the single-breath determination of carbon monoxide uptake in the lung.

Authors:  N Macintyre; R O Crapo; G Viegi; D C Johnson; C P M van der Grinten; V Brusasco; F Burgos; R Casaburi; A Coates; P Enright; P Gustafsson; J Hankinson; R Jensen; R McKay; M R Miller; D Navajas; O F Pedersen; R Pellegrino; J Wanger
Journal:  Eur Respir J       Date:  2005-10       Impact factor: 16.671

4.  Standardisation of spirometry.

Authors:  M R Miller; J Hankinson; V Brusasco; F Burgos; R Casaburi; A Coates; R Crapo; P Enright; C P M van der Grinten; P Gustafsson; R Jensen; D C Johnson; N MacIntyre; R McKay; D Navajas; O F Pedersen; R Pellegrino; G Viegi; J Wanger
Journal:  Eur Respir J       Date:  2005-08       Impact factor: 16.671

5.  Immunohistochemical study of matrix metalloproteinases (MMPs) and their tissue inhibitors (TIMPs) in pulmonary lymphangioleiomyomatosis (LAM).

Authors:  T Hayashi; M V Fleming; W G Stetler-Stevenson; L A Liotta; J Moss; V J Ferrans; W D Travis
Journal:  Hum Pathol       Date:  1997-09       Impact factor: 3.466

6.  Decline in lung function in patients with lymphangioleiomyomatosis treated with or without progesterone.

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Journal:  Chest       Date:  2004-12       Impact factor: 9.410

7.  Comprehensive evaluation of 35 patients with lymphangioleiomyomatosis.

Authors:  S C Chu; K Horiba; J Usuki; N A Avila; C C Chen; W D Travis; V J Ferrans; J Moss
Journal:  Chest       Date:  1999-04       Impact factor: 9.410

8.  Pulmonary lymphangioleiomyomatosis. A study of 69 patients. Groupe d'Etudes et de Recherche sur les Maladies "Orphelines" Pulmonaires (GERM"O"P).

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9.  Acute bronchodilator response has limited value in differentiating bronchial asthma from COPD.

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10.  Pulmonary lymphangioleiomyomatosis: a report of 46 patients including a clinicopathologic study of prognostic factors.

Authors:  M Kitaichi; K Nishimura; H Itoh; T Izumi
Journal:  Am J Respir Crit Care Med       Date:  1995-02       Impact factor: 21.405

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  13 in total

Review 1.  Diffuse Cystic Lung Disease. Part I.

Authors:  Nishant Gupta; Robert Vassallo; Kathryn A Wikenheiser-Brokamp; Francis X McCormack
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2.  Analysis of the MILES cohort reveals determinants of disease progression and treatment response in lymphangioleiomyomatosis.

Authors:  Nishant Gupta; Hye-Seung Lee; Lisa R Young; Charlie Strange; Joel Moss; Lianne G Singer; Koh Nakata; Alan F Barker; Jeffrey T Chapman; Mark L Brantly; James M Stocks; Kevin K Brown; Joseph P Lynch; Hilary J Goldberg; Gregory P Downey; Angelo M Taveira-DaSilva; Jeffrey P Krischer; Kenneth Setchell; Bruce C Trapnell; Yoshikazu Inoue; Francis X McCormack
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3.  Involvement of lymphatics in lymphangioleiomyomatosis.

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Review 4.  Lymphangioleiomyomatosis: what do we know and what are we looking for?

Authors:  S Harari; O Torre; J Moss
Journal:  Eur Respir Rev       Date:  2011-03

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6.  Clinical predictors of mortality and cause of death in lymphangioleiomyomatosis: a population-based registry.

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9.  Is sirolimus a therapeutic option for patients with progressive pulmonary lymphangioleiomyomatosis?

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Review 10.  Clinical features, epidemiology, and therapy of lymphangioleiomyomatosis.

Authors:  Angelo M Taveira-DaSilva; Joel Moss
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