Literature DB >> 19434929

Cellular immune responses during high-dose interferon-alpha induction therapy for hepatitis C virus infection.

Eleanor Barnes1, Huub C Gelderblom, Isla Humphreys, Nasser Semmo, Henk W Reesink, Marcel G H M Beld, René A W van Lier, Paul Klenerman.   

Abstract

BACKGROUND: The effect that high-dose interferon (IFN)-alpha induction therapy for hepatitis C virus (HCV) infection has on cellular immune responses is currently unknown.
METHODS: Thirty-one treatment-naive patients with chronic HCV infection received amantadine and ribavirin, combined with 6 weeks of high-dose IFN-alpha-2b induction therapy followed by weekly pegylated IFN-alpha-2b, for 24 or 48 weeks. Using IFN-gamma and interleukin (IL)-2 enzyme-linked immunospot (ELISpot) assays, we analyzed the pattern of cytokine secretion by structural and nonstructural HCV- and cytomegalovirus (CMV)-specific T cells before, during, and after therapy.
RESULTS: HCV-specific T cell responses, which were predominantly IFN-gamma secreting and which correlated with alanine transaminase levels (r2 = 0.45; P = .001), were found before treatment in 10 of 15 patients with a sustained virological response (SVR) and in 11 of 16 in the non-SVR group. There was a striking loss of IFN-gamma and IL-2 HCV-specific T cells during therapy, predominantly in the SVR group. This response recovered after cessation of therapy, regardless of outcome. Suppression of CMV-specific T cell responses, in addition to total lymphocyte counts, was also observed.
CONCLUSIONS: High-dose IFN-alpha induction therapy leads to a profound decline in IL-2- and IFN-gamma-secreting HCV- and CMV-specific T cells. These data indicate that restoration of T cell responses is unlikely to be causally linked to an early response or SVR to therapy.

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Year:  2009        PMID: 19434929     DOI: 10.1086/597072

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  23 in total

Review 1.  Hepatitis C virus-specific cytotoxic T cell response restoration after treatment-induced hepatitis C virus control.

Authors:  Juan-Ramón Larrubia; Elia Moreno-Cubero; Joaquín Miquel; Eduardo Sanz-de-Villalobos
Journal:  World J Gastroenterol       Date:  2015-03-28       Impact factor: 5.742

Review 2.  Immune and non-immune responses to hepatitis C virus infection.

Authors:  Jiaren Sun; Ricardo Rajsbaum; MinKyung Yi
Journal:  World J Gastroenterol       Date:  2015-10-14       Impact factor: 5.742

3.  HCV-specific immune responses induced by CIGB-230 in combination with IFN-α plus ribavirin.

Authors:  Yalena Amador-Cañizares; Gillian Martínez-Donato; Liz Alvarez-Lajonchere; Claudia Vasallo; Mariacarla Dausá; Daylen Aguilar-Noriega; Carmen Valenzuela; Ivette Raíces; Jean Dubuisson; Czeslaw Wychowski; Zurina Cinza-Estévez; Marlén Castellanos; Magdalys Núñez; Anny Armas; Yaimé González; Ismariley Revé; Ivis Guerra; Angel Pérez Aguiar; Santiago Dueñas-Carrera
Journal:  World J Gastroenterol       Date:  2014-01-07       Impact factor: 5.742

4.  Memory T Cell Proliferation before Hepatitis C Virus Therapy Predicts Antiviral Immune Responses and Treatment Success.

Authors:  Gema Méndez-Lagares; Ding Lu; Connie Chen; Norah Terrault; Mark R Segal; Mandana Khalili; Alexander Monto; Hui Shen; M Michele Manos; Lewis L Lanier; James C Ryan; Joseph M McCune; Dennis J Hartigan-O'Connor
Journal:  J Immunol       Date:  2017-12-20       Impact factor: 5.422

5.  Could a loss of memory T cells limit responses to hepatitis C virus (HCV) antigens in blood leucocytes from patients chronically infected with HCV before and during pegylated interferon-alpha and ribavirin therapy?

Authors:  S Lee; T Hammond; M W Watson; J P Flexman; W Cheng; S Fernandez; P Price
Journal:  Clin Exp Immunol       Date:  2010-04-09       Impact factor: 4.330

Review 6.  Current progress in host innate and adaptive immunity against hepatitis C virus infection.

Authors:  Jijing Shi; Yuanyuan Li; Wenxian Chang; Xuexiu Zhang; Fu-Sheng Wang
Journal:  Hepatol Int       Date:  2017-06-22       Impact factor: 6.047

7.  Monocyte activation by interferon α is associated with failure to achieve a sustained virologic response after treatment for hepatitis C virus infection.

Authors:  Dennis J Hartigan-O'Connor; Din Lin; James C Ryan; Valentina A Shvachko; Myrna L Cozen; Mark R Segal; Norah A Terrault; Lewis L Lanier; M Michele Manos; Joseph M McCune
Journal:  J Infect Dis       Date:  2013-12-10       Impact factor: 5.226

Review 8.  Vaccination for hepatitis C virus: closing in on an evasive target.

Authors:  John Halliday; Paul Klenerman; Eleanor Barnes
Journal:  Expert Rev Vaccines       Date:  2011-05       Impact factor: 5.217

9.  Broadly directed virus-specific CD4+ T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence.

Authors:  Julian Schulze Zur Wiesch; Donatella Ciuffreda; Lia Lewis-Ximenez; Victoria Kasprowicz; Brian E Nolan; Hendrik Streeck; Jasneet Aneja; Laura L Reyor; Todd M Allen; Ansgar W Lohse; Barbara McGovern; Raymond T Chung; William W Kwok; Arthur Y Kim; Georg M Lauer
Journal:  J Exp Med       Date:  2012-01-02       Impact factor: 14.307

10.  Clinical testing of a dendritic cell targeted therapeutic vaccine in patients with chronic hepatitis C virus infection.

Authors:  Aintzane Zabaleta; Delia D'Avola; Itziar Echeverria; Diana Llopiz; Leyre Silva; Lorea Villanueva; José Ignacio Riezu-Boj; Esther Larrea; Alexander Pereboev; Juan José Lasarte; Iago Rodriguez-Lago; Mercedes Iñarrairaegui; Bruno Sangro; Jesús Prieto; Pablo Sarobe
Journal:  Mol Ther Methods Clin Dev       Date:  2015-03-11       Impact factor: 6.698

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