Literature DB >> 19424025

Muscular dystrophy therapy by nonautologous mesenchymal stem cells: muscle regeneration without immunosuppression and inflammation.

Arsalan Shabbir1, David Zisa, Merced Leiker, Curtis Johnston, Huey Lin, Techung Lee.   

Abstract

BACKGROUND: The use of nonautologous stem cells isolated from healthy donors for stem-cell therapy is an attractive approach, because the stem cells can be culture expanded in advance, thoroughly tested, and formulated into off-the-shelf medicine. However, human leukocyte antigen compatibility and related immunosuppressive protocols can compromise therapeutic efficacy and cause unwanted side effects.
METHODS: Mesenchymal stem cells (MSCs) have been postulated to possess unique immune regulatory function. We explored the immunomodulatory property of human and porcine MSCs for the treatment of delta-sarcoglycan-deficient dystrophic hamster muscle without immunosuppression. Circulating and tissue markers of inflammation were analyzed. Muscle regeneration and stem-cell fate were characterized.
RESULTS: Total white blood cell counts and leukocyte-distribution profiles were similar among the saline- and MSC-injected dystrophic hamsters 1 month posttreatment. Circulating levels of immunoglobulin A, vascular cell adhesion molecule-1, myeloperoxidase, and major cytokines involved in inflammatory response were not elevated by MSCs, nor were expression of the leukocyte common antigen CD45 and the cytokine transcriptional activator NF-kappaB in the injected muscle. Treated muscles exhibited increased cell-cycle activity and attenuated oxidative stress. Injected MSCs were found to be trapped in the musculature, contribute to both preexisting and new muscle fibers, and mediates capillary formation.
CONCLUSIONS: Intramuscular injection of nonautologous MSCs can be safely used for the treatment of dystrophic muscle in immunocompetent hosts without inflaming the host immune system.

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Year:  2009        PMID: 19424025      PMCID: PMC2746453          DOI: 10.1097/TP.0b013e3181a1719b

Source DB:  PubMed          Journal:  Transplantation        ISSN: 0041-1337            Impact factor:   4.939


  42 in total

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5.  Dystrophin mutations predict cellular susceptibility to oxidative stress.

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Journal:  Muscle Nerve       Date:  2000-05       Impact factor: 3.217

6.  Adeno-associated virus vector-mediated gene transfer into dystrophin-deficient skeletal muscles evokes enhanced immune response against the transgene product.

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7.  Suppression of allogeneic T-cell proliferation by human marrow stromal cells: implications in transplantation.

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8.  Gene expression profile of mouse bone marrow stromal cells determined by cDNA microarray analysis.

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  33 in total

1.  Intramuscular VEGF activates an SDF1-dependent progenitor cell cascade and an SDF1-independent muscle paracrine cascade for cardiac repair.

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2.  Photoacoustic imaging of mesenchymal stem cells in living mice via silica-coated gold nanorods.

Authors:  Jesse V Jokerst; Mridhula Thangaraj; Paul J Kempen; Robert Sinclair; Sanjiv S Gambhir
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3.  Activation of host tissue trophic factors through JAK-STAT3 signaling: a mechanism of mesenchymal stem cell-mediated cardiac repair.

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Journal:  Am J Physiol Heart Circ Physiol       Date:  2010-09-17       Impact factor: 4.733

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Review 8.  Do mesenchymal stem cells function across species barriers? Relevance for xenotransplantation.

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9.  Activation of Toll-like receptor 3 amplifies mesenchymal stem cell trophic factors and enhances therapeutic potency.

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10.  Vascular endothelial growth factor (VEGF) as a key therapeutic trophic factor in bone marrow mesenchymal stem cell-mediated cardiac repair.

Authors:  David Zisa; Arsalan Shabbir; Gen Suzuki; Techung Lee
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