Literature DB >> 19402387

Comparative genomic hybridization-based oncogenetic tree model for genetic classification of breast cancer.

Katrin Friedrich1, Anja von Heydebreck, Gunter Haroske, Jens Scheithauer, Wolfdietrich Meyer, Klaus Dietmar Kunze, Gustavo Baretton.   

Abstract

OBJECTIVE: To describe a genetic progression pathway in breast cancer by a maximum likelihood-based tree model representing the dependencies between chromosomal imbalances. STUDY
DESIGN: One hundred six cases were studied by comparative genomic hybridization, followed by maximum likelihood estimation of an oncogenetic tree model.
RESULTS: The tree model identified 3 clusters with correlated chromosomal imbalances. The first cluster included losses at 4q, 5q, 6q, 9p, 13q and a gain at 17q; the second cluster included gains at 1q, 8q, 16p and 20q; the third cluster included losses at 8p, 11q, 16q and 18q. The imbalances nearest the root of the tree were the loss at 13q (cluster 1), the gain at 1q (cluster 2) and the loss at 18q (cluster 3), reflecting an early change in breast cancer evolution. Cox regression analysis revealed the tumor stage and the grade as relevant for overall survival (p = 0.001) and the tumor stage, the grade and the loss at 16q as relevant for disease-free survival (p = 0.001).
CONCLUSION: Methods like oncogenetic tree analysis provide insights into the genetic progression of breast cancer and may extract relevant markers detected by screening methods like comparative genomic hybridization for further studies.

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Year:  2009        PMID: 19402387

Source DB:  PubMed          Journal:  Anal Quant Cytol Histol        ISSN: 0884-6812            Impact factor:   0.302


  5 in total

1.  Spontaneous transformation of murine epithelial cells requires the early acquisition of specific chromosomal aneuploidies and genomic imbalances.

Authors:  Hesed M Padilla-Nash; Karen Hathcock; Nicole E McNeil; David Mack; Daniel Hoeppner; Rea Ravin; Turid Knutsen; Raluca Yonescu; Danny Wangsa; Kathleen Dorritie; Linda Barenboim; Yue Hu; Thomas Ried
Journal:  Genes Chromosomes Cancer       Date:  2011-12-08       Impact factor: 5.006

2.  Copy number imbalances between screen- and symptom-detected breast cancers and impact on disease-free survival.

Authors:  A M Brewster; P Thompson; A A Sahin; K Do; M Edgerton; J L Murray; S Tsavachidis; R Zhou; Y Liu; L Zhang; G Mills; M Bondy
Journal:  Cancer Prev Res (Phila)       Date:  2011-07-27

3.  Chromosome 1q25.3 copy number alterations in primary breast cancers detected by multiplex ligation-dependent probe amplification and allelic imbalance assays and its comparison with fluorescent in situ hybridization assays.

Authors:  Emilia Wiechec; Jens Overgaard; Eigil Kjeldsen; Lise Lotte Hansen
Journal:  Cell Oncol (Dordr)       Date:  2012-12-18       Impact factor: 6.730

4.  A mathematical methodology for determining the temporal order of pathway alterations arising during gliomagenesis.

Authors:  Yu-Kang Cheng; Rameen Beroukhim; Ross L Levine; Ingo K Mellinghoff; Eric C Holland; Franziska Michor
Journal:  PLoS Comput Biol       Date:  2012-01-05       Impact factor: 4.475

5.  The role of KIF14 in patient-derived primary cultures of high-grade serous ovarian cancer cells.

Authors:  Brigitte L Thériault; Paulina Cybulska; Patricia A Shaw; Brenda L Gallie; Marcus Q Bernardini
Journal:  J Ovarian Res       Date:  2014-12-21       Impact factor: 4.234

  5 in total

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