Literature DB >> 19398086

Increased high mobility group box-1 protein levels are associated with impaired cardiopulmonary and echocardiographic findings after acute myocardial infarction.

Plinio Cirillo1, Francesco Giallauria, Mario Pacileo, Gianluca Petrillo, Mariantonietta D'Agostino, Carlo Vigorito, Massimo Chiariello.   

Abstract

BACKGROUND: Several markers of systemic inflammation seem to play an active role in the pathophysiology of acute coronary syndrome and its evolution. High mobility group box-1 (HMGB-1), a ubiquitous nuclear protein constitutively expressed in quiescent cells, was recently recognized as a newer critical mediator of inflammatory diseases. The present study aimed to evaluate the possible association between HMGB-1 levels and structural and functional indices of cardiovascular performance such as cardiopulmonary and Doppler-echocardiography indices in patients after acute myocardial infarction (MI). METHODS AND
RESULTS: Fifty-four consecutive patients (mean age 58.3 years, 83% males) recovering from acute MI were included in the study protocol. All patients underwent Doppler-echocardiography, cardiopulmonary exercise, and HMGB-1 assay. HMGB-1 levels in acute MI patients were significantly higher compared with age- and body mass index-matched controls (14.8 +/- 6.8 vs. 2.3 +/- 1.0 ng/mL, P < .0001, respectively). Postinfarction patients showed oxygen consumption at peak exercise (VO(2 peak)) = 14.4 +/- 4.2 mL x kg x min and a slope of increase in ventilation over carbon dioxide output (VE/VCO(2 slope)) = 32.1 +/- 6.2, whereas Doppler-echocardiography values were: left ventricular end-diastolic volume (LVEDV) = 53.4 +/- 8.2 mL/m(2); left ventricular ejection fraction (LVEF) = 41.7 +/- 7.0%. Multiple linear regression analysis (stepwise method) showed that VO(2 peak) (beta = -0.276, P = .012), VE/VCO(2 slope) (beta = 0.244, P = .005), LVEDV (beta = 0.267, P = .018), peak creatine kinase-MB (beta = 0.339, P = .004), peak Troponin I (beta = 0.244, P = .002), and LVEF (beta = -0.312, P = .021) were significantly associated with HMGB-1 levels.
CONCLUSIONS: The present study demonstrated that in postinfarction patients, HMGB-1 levels were significantly higher compared with controls, and significantly correlated with cardiopulmonary and Doppler-echocardiography parameters.

Entities:  

Mesh:

Substances:

Year:  2009        PMID: 19398086     DOI: 10.1016/j.cardfail.2008.11.010

Source DB:  PubMed          Journal:  J Card Fail        ISSN: 1071-9164            Impact factor:   5.712


  11 in total

1.  Apoptosis of hematopoietic progenitor-derived adipose tissue-resident macrophages contributes to insulin resistance after myocardial infarction.

Authors:  Sathish Babu Vasamsetti; Emilie Coppin; Xinyi Zhang; Jonathan Florentin; Sasha Koul; Matthias Götberg; Andrew S Clugston; Floyd Thoma; John Sembrat; Grant C Bullock; Dennis Kostka; Claudette M St Croix; Ansuman Chattopadhyay; Mauricio Rojas; Suresh R Mulukutla; Partha Dutta
Journal:  Sci Transl Med       Date:  2020-07-22       Impact factor: 17.956

Review 2.  The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.

Authors:  Sumanth D Prabhu; Nikolaos G Frangogiannis
Journal:  Circ Res       Date:  2016-06-24       Impact factor: 17.367

3.  Autonomic dysfunction is associated with high mobility group box-1 levels in patients after acute myocardial infarction.

Authors:  Francesco Giallauria; Plinio Cirillo; Rosa Lucci; Mario Pacileo; Mariantonietta D'Agostino; Paola Maietta; Alessandra Vitelli; Massimo Chiariello; Carlo Vigorito
Journal:  Atherosclerosis       Date:  2009-07-14       Impact factor: 5.162

Review 4.  HMGB1 in health and disease.

Authors:  Rui Kang; Ruochan Chen; Qiuhong Zhang; Wen Hou; Sha Wu; Lizhi Cao; Jin Huang; Yan Yu; Xue-Gong Fan; Zhengwen Yan; Xiaofang Sun; Haichao Wang; Qingde Wang; Allan Tsung; Timothy R Billiar; Herbert J Zeh; Michael T Lotze; Daolin Tang
Journal:  Mol Aspects Med       Date:  2014-07-08

5.  Angiotensin-converting enzyme 2 inhibits high-mobility group box 1 and attenuates cardiac dysfunction post-myocardial ischemia.

Authors:  Yan Fei Qi; Juan Zhang; Lei Wang; Vinayak Shenoy; Eric Krause; S Paul Oh; Carl J Pepine; Michael J Katovich; Mohan K Raizada
Journal:  J Mol Med (Berl)       Date:  2016-01       Impact factor: 4.599

Review 6.  Clinical Evidence for Q10 Coenzyme Supplementation in Heart Failure: From Energetics to Functional Improvement.

Authors:  Anna Di Lorenzo; Gabriella Iannuzzo; Alessandro Parlato; Gianluigi Cuomo; Crescenzo Testa; Marta Coppola; Giuseppe D'Ambrosio; Domenico Alessandro Oliviero; Silvia Sarullo; Giuseppe Vitale; Cinzia Nugara; Filippo M Sarullo; Francesco Giallauria
Journal:  J Clin Med       Date:  2020-04-27       Impact factor: 4.241

7.  Role of HMGB1/TLR4 Axis in Ischemia/Reperfusion-Impaired Extracellular Glutamate Clearance in Primary Astrocytes.

Authors:  Chia-Ho Lin; Han-Yu Chen; Kai-Che Wei
Journal:  Cells       Date:  2020-12-03       Impact factor: 6.600

Review 8.  Danger signals in the initiation of the inflammatory response after myocardial infarction.

Authors:  J J de Haan; M B Smeets; G Pasterkamp; F Arslan
Journal:  Mediators Inflamm       Date:  2013-11-30       Impact factor: 4.711

9.  Prognostic role of serum high mobility group box 1 concentration in cardiac surgery.

Authors:  Namo Kim; Sak Lee; Jeong-Rim Lee; Young-Lan Kwak; Ji-Hae Jun; Jae-Kwang Shim
Journal:  Sci Rep       Date:  2020-04-14       Impact factor: 4.379

Review 10.  Oncology and Cardiac Rehabilitation: An Underrated Relationship.

Authors:  E Venturini; G Iannuzzo; A D'Andrea; M Pacileo; L Tarantini; M L Canale; M Gentile; G Vitale; F M Sarullo; R Vastarella; A Di Lorenzo; C Testa; A Parlato; C Vigorito; F Giallauria
Journal:  J Clin Med       Date:  2020-06-10       Impact factor: 4.964

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.