Literature DB >> 19372949

Treating HIV/AIDS by reducing immune system activation: the paradox of immune deficiency and immune hyperactivation.

Franco Lori1.   

Abstract

PURPOSE OF REVIEW: To collect published evidence in support of a novel immune therapeutic approach to reduce the excess of immune activation that ultimately turns into immune deficiency in HIV/AIDS. RECENT
FINDINGS: A large body of evidence has been collected in support of the pathogenetic interpretation that prolonged immune overactivation induced by HIV during the course of chronic infection exhausts the immune system and leads to AIDS. Some groups are exploring the possibility of therapeutic interventions to limit the immune system overload. Cytostatic drugs appear promising candidates to achieve the goal, as they restrain cell proliferation without blocking it. At the same time, they do not affect cellular response to antigenic stimulation, and at appropriate dosages are not immune suppressive.
SUMMARY: Presently available antiretrovirals only partially reduce immune system activation during chronic HIV infection. Further clinical research is warranted to test cytostatic drugs to avert immune overactivation and prevent progression to AIDS.

Entities:  

Year:  2008        PMID: 19372949     DOI: 10.1097/COH.0b013e3282f525cf

Source DB:  PubMed          Journal:  Curr Opin HIV AIDS        ISSN: 1746-630X            Impact factor:   4.283


  5 in total

1.  Minocycline suppresses activation of nuclear factor of activated T cells 1 (NFAT1) in human CD4+ T cells.

Authors:  Gregory L Szeto; Joel L Pomerantz; David R M Graham; Janice E Clements
Journal:  J Biol Chem       Date:  2011-01-31       Impact factor: 5.157

Review 2.  Hypothesis: bacteria control host appetites.

Authors:  Vic Norris; Franck Molina; Andrew T Gewirtz
Journal:  J Bacteriol       Date:  2012-11-09       Impact factor: 3.490

3.  Immune activation in the pathogenesis of treated chronic HIV disease: a workshop summary.

Authors:  Susan F Plaeger; Brenda S Collins; Runa Musib; Steven G Deeks; Sarah Read; Alan Embry
Journal:  AIDS Res Hum Retroviruses       Date:  2011-09-27       Impact factor: 2.205

4.  The immunosuppressive role of IL-32 in lymphatic tissue during HIV-1 infection.

Authors:  Anthony J Smith; Chad M Toledo; Stephen W Wietgrefe; Lijie Duan; Timothy W Schacker; Cavan S Reilly; Ashley T Haase
Journal:  J Immunol       Date:  2011-04-27       Impact factor: 5.422

Review 5.  HIV as a Cause of Immune Activation and Immunosenescence.

Authors:  T Sokoya; H C Steel; M Nieuwoudt; T M Rossouw
Journal:  Mediators Inflamm       Date:  2017-10-25       Impact factor: 4.711

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.