Literature DB >> 19334058

Selective vulnerability in Alzheimer's disease: amyloid precursor protein and p75(NTR) interaction.

Joanna Fombonne1, Shahrooz Rabizadeh, Surita Banwait, Patrick Mehlen, Dale E Bredesen.   

Abstract

OBJECTIVE: Selective neuronal vulnerability in neurodegenerative diseases is poorly understood. In Alzheimer's disease, the basal forebrain cholinergic neurons are selectively vulnerable, putatively because of their expression of the cell death mediator p75(NTR) (the common neurotrophin receptor), and its interaction with proapoptotic ligands pro-nerve growth factor and amyloid-beta peptide. However, the relation between amyloid precursor protein (APP) and p75(NTR) has not been described previously.
METHODS: APP and p75(NTR) were assayed for interaction by coimmunoprecipitation in vitro and in vivo, yeast two-hybrid assay, bioluminescence resonance energy transfer, and confocal microscopy. Effects on APP processing and signaling were studied using immunoblotting, enzyme-linked immunosorbent assays, and luciferase reporter assays.
RESULTS: The results of this study are as follows: (1) p75(NTR) and APP interact directly; (2) this interaction is modified by ligands nerve growth factor and beta-amyloid; (3) APP and p75(NTR) colocalization in vivo is modified in Alzheimer's model transgenic mice; (4) APP processing is altered by p75(NTR), and to a lesser extent, p75(NTR) processing is altered by the presence of APP; (5) APP-dependent transcription mediated by Fe65 is blocked by p75(NTR); and (6) coexpression of APP and p75(NTR) triggers cell death.
INTERPRETATION: These results provide new insight into the emerging signaling network that mediates the Alzheimer's phenotype and into the mechanism of basal forebrain cholinergic neuronal selective vulnerability. In addition, the results argue that the interaction between APP and p75(NTR) may represent a therapeutic target in Alzheimer's disease.

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Year:  2009        PMID: 19334058      PMCID: PMC3057030          DOI: 10.1002/ana.21578

Source DB:  PubMed          Journal:  Ann Neurol        ISSN: 0364-5134            Impact factor:   10.422


  29 in total

1.  A second cytotoxic proteolytic peptide derived from amyloid beta-protein precursor.

Authors:  D C Lu; S Rabizadeh; S Chandra; R F Shayya; L M Ellerby; X Ye; G S Salvesen; E H Koo; D E Bredesen
Journal:  Nat Med       Date:  2000-04       Impact factor: 53.440

2.  Abeta induces cell death by direct interaction with its cognate extracellular domain on APP (APP 597-624).

Authors:  G M Shaked; M P Kummer; D C Lu; V Galvan; D E Bredesen; E H Koo
Journal:  FASEB J       Date:  2006-04-24       Impact factor: 5.191

3.  Fe65 stimulates proteolytic liberation of the beta-amyloid precursor protein intracellular domain.

Authors:  Jesse C Wiley; Elise A Smith; Mark P Hudson; Warren C Ladiges; Mark Bothwell
Journal:  J Biol Chem       Date:  2007-09-11       Impact factor: 5.157

4.  Deficits in synaptic transmission and learning in amyloid precursor protein (APP) transgenic mice require C-terminal cleavage of APP.

Authors:  Michael J Saganich; Brock E Schroeder; Veronica Galvan; Dale E Bredesen; Edward H Koo; Stephen F Heinemann
Journal:  J Neurosci       Date:  2006-12-27       Impact factor: 6.167

5.  Pro-NGF isolated from the human brain affected by Alzheimer's disease induces neuronal apoptosis mediated by p75NTR.

Authors:  Carlos E Pedraza; Petar Podlesniy; Noemí Vidal; Juan Carlos Arévalo; Ramee Lee; Barbara Hempstead; Isidre Ferrer; Montse Iglesias; Carme Espinet
Journal:  Am J Pathol       Date:  2005-02       Impact factor: 4.307

Review 6.  Neurotrophin receptor-based strategies for Alzheimer's disease.

Authors:  Frank M Longo; Stephen M Massa
Journal:  Curr Alzheimer Res       Date:  2005-04       Impact factor: 3.498

7.  Signal transduction in Alzheimer disease: p21-activated kinase signaling requires C-terminal cleavage of APP at Asp664.

Authors:  Thuy-Vi V Nguyen; Veronica Galvan; Wei Huang; Surita Banwait; Huidong Tang; Junli Zhang; Dale E Bredesen
Journal:  J Neurochem       Date:  2007-11-06       Impact factor: 5.372

8.  A cyclic peptide that binds p75(NTR) protects neurones from beta amyloid (1-40)-induced cell death.

Authors:  M Yaar; S Zhai; I Panova; R E Fine; P B Eisenhauer; J K Blusztajn; I Lopez-Coviella; B A Gilchrest
Journal:  Neuropathol Appl Neurobiol       Date:  2007-06-27       Impact factor: 8.090

9.  Long-term prevention of Alzheimer's disease-like behavioral deficits in PDAPP mice carrying a mutation in Asp664.

Authors:  Veronica Galvan; Junli Zhang; Olivia F Gorostiza; Surita Banwait; Wei Huang; Marina Ataie; Huidong Tang; Dale E Bredesen
Journal:  Behav Brain Res       Date:  2008-04-08       Impact factor: 3.332

10.  TgCRND8 amyloid precursor protein transgenic mice exhibit an altered gamma-secretase processing and an aggressive, additive amyloid pathology subject to immunotherapeutic modulation.

Authors:  Gregory D Van Vickle; Chera L Esh; Walter M Kalback; R Lyle Patton; Dean C Luehrs; Tyler A Kokjohn; Frederick G Fifield; Paul E Fraser; David Westaway; Joanne McLaurin; John Lopez; Daniel Brune; Amanda J Newel; Marissa Poston; Thomas G Beach; Alex E Roher
Journal:  Biochemistry       Date:  2007-08-18       Impact factor: 3.162

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  31 in total

Review 1.  Roles of amyloid precursor protein family members in neuroprotection, stress signaling and aging.

Authors:  Donat Kögel; Thomas Deller; Christian Behl
Journal:  Exp Brain Res       Date:  2011-11-16       Impact factor: 1.972

Review 2.  Cellular Trafficking of Amyloid Precursor Protein in Amyloidogenesis Physiological and Pathological Significance.

Authors:  Noralyn Basco Mañucat-Tan; Khalil Saadipour; Yan-Jiang Wang; Larisa Bobrovskaya; Xin-Fu Zhou
Journal:  Mol Neurobiol       Date:  2018-05-24       Impact factor: 5.590

Review 3.  Restoring Soluble Amyloid Precursor Protein α Functions as a Potential Treatment for Alzheimer's Disease.

Authors:  Ahsan Habib; Darrell Sawmiller; Jun Tan
Journal:  J Neurosci Res       Date:  2016-08-17       Impact factor: 4.164

4.  CA1 pyramidal neuron gene expression mosaics in the Ts65Dn murine model of Down syndrome and Alzheimer's disease following maternal choline supplementation.

Authors:  Melissa J Alldred; Helen M Chao; Sang Han Lee; Judah Beilin; Brian E Powers; Eva Petkova; Barbara J Strupp; Stephen D Ginsberg
Journal:  Hippocampus       Date:  2018-02-12       Impact factor: 3.899

5.  Resilience of precuneus neurotrophic signaling pathways despite amyloid pathology in prodromal Alzheimer's disease.

Authors:  Sylvia E Perez; Bin He; Muhammad Nadeem; Joanne Wuu; Stephen W Scheff; Eric E Abrahamson; Milos D Ikonomovic; Elliott J Mufson
Journal:  Biol Psychiatry       Date:  2014-01-11       Impact factor: 13.382

Review 6.  Recent advances in our understanding of neurodegeneration.

Authors:  Kurt A Jellinger
Journal:  J Neural Transm (Vienna)       Date:  2009-06-05       Impact factor: 3.575

7.  The effects of transmembrane sequence and dimerization on cleavage of the p75 neurotrophin receptor by γ-secretase.

Authors:  Alex M Sykes; Nickless Palstra; Daniel Abankwa; Justine M Hill; Sune Skeldal; Dusan Matusica; Prahatha Venkatraman; John F Hancock; Elizabeth J Coulson
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Review 8.  One protein, multiple pathologies: multifaceted involvement of amyloid β in neurodegenerative disorders of the brain and retina.

Authors:  Vivek Gupta; Veer B Gupta; Nitin Chitranshi; Sumudu Gangoda; Roshana Vander Wall; Mojdeh Abbasi; Mojtaba Golzan; Yogita Dheer; Tejal Shah; Alberto Avolio; Roger Chung; Ralph Martins; Stuart Graham
Journal:  Cell Mol Life Sci       Date:  2016-06-22       Impact factor: 9.261

9.  Small molecule p75NTR ligands reduce pathological phosphorylation and misfolding of tau, inflammatory changes, cholinergic degeneration, and cognitive deficits in AβPP(L/S) transgenic mice.

Authors:  Thuy-Vi V Nguyen; Lin Shen; Lilith Vander Griend; Lisa N Quach; Nadia P Belichenko; Nay Saw; Tao Yang; Mehrdad Shamloo; Tony Wyss-Coray; Stephen M Massa; Frank M Longo
Journal:  J Alzheimers Dis       Date:  2014       Impact factor: 4.472

10.  Neurodegeneration in Alzheimer's disease: caspases and synaptic element interdependence.

Authors:  Dale E Bredesen
Journal:  Mol Neurodegener       Date:  2009-06-26       Impact factor: 14.195

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