| Literature DB >> 19251657 |
Mohammad Alimohammadi1, Noémie Dubois, Filip Sköldberg, Asa Hallgren, Isabelle Tardivel, Håkan Hedstrand, Jan Haavik, Eystein S Husebye, Jan Gustafsson, Fredrik Rorsman, Antonella Meloni, Christer Janson, Bernard Vialettes, Merja Kajosaari, William Egner, Ravishankar Sargur, Fredrik Pontén, Zahir Amoura, Alain Grimfeld, Filippo De Luca, Corrado Betterle, Jaakko Perheentupa, Olle Kämpe, Jean-Claude Carel.
Abstract
Patients with autoimmune polyendocrine syndrome type 1 (APS-1) suffer from multiple organ-specific autoimmunity with autoantibodies against target tissue-specific autoantigens. Endocrine and nonendocrine organs such as skin, hair follicles, and liver are targeted by the immune system. Despite sporadic observations of pulmonary symptoms among APS-1 patients, an autoimmune mechanism for pulmonary involvement has not been elucidated. We report here on a subset of APS-1 patients with respiratory symptoms. Eight patients with pulmonary involvement were identified. Severe airway obstruction was found in 4 patients, leading to death in 2. Immunoscreening of a cDNA library using serum samples from a patient with APS-1 and obstructive respiratory symptoms identified a putative potassium channel regulator (KCNRG) as a pulmonary autoantigen. Reactivity to recombinant KCNRG was assessed in 110 APS-1 patients by using immunoprecipitation. Autoantibodies to KCNRG were present in 7 of the 8 patients with respiratory symptoms, but in only 1 of 102 APS-1 patients without respiratory symptoms. Expression of KCNRG messenger RNA and protein was found to be predominantly restricted to the epithelial cells of terminal bronchioles. Autoantibodies to KCNRG, a protein mainly expressed in bronchial epithelium, are strongly associated with pulmonary involvement in APS-1. These findings may facilitate the recognition, diagnosis, characterization, and understanding of the pulmonary manifestations of APS-1.Entities:
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Year: 2009 PMID: 19251657 PMCID: PMC2648890 DOI: 10.1073/pnas.0809986106
Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN: 0027-8424 Impact factor: 11.205