Literature DB >> 19246480

Premature death in adults with 22q11.2 deletion syndrome.

A S Bassett1, E W C Chow, J Husted, K A Hodgkinson, E Oechslin, L Harris, C Silversides.   

Abstract

BACKGROUND: 22q11.2 deletion syndrome (22q11.2DS) is a multisystem disease with a prevalence of 1/4000. Variable expression of congenital and later onset features contributes to its under-recognition. Longevity in those surviving childhood is believed to be normal but data are limited.
METHODS: We prospectively followed 264 subjects; 102 adults (>17 years) with 22q11.2DS (44 male (M), 58 female (F); mean (SD) age 33.6 (10.9) years) and their 162 unaffected siblings (77 M, 85 F; mean age 36.1 (12.2) years). We compared survival between groups using Kaplan-Meier estimates.
RESULTS: Twelve (11.8%; 4 M, 8 F) individuals with 22q11.2DS and no siblings died (p<0.0001). Survival to ages 40 and 50 years was 89.9% and 73.9%, respectively. Median age at death was 41.5 (range 18.1-68.6) years. Deaths included two (7.7%) of 26 subjects with neither major congenital heart disease (CHD) nor schizophrenia. Four of six sudden and unexpected deaths occurred in individuals with no major CHD. There was no evidence of cancer or coronary artery disease or family history of sudden death in the 12 patients who died, six of whom had autopsies. DISCUSSION: Individuals with 22q11.2DS who survive childhood have diminished life expectancy and increased risk of sudden death not attributable to any single factor. Some sudden and/or premature deaths observed in the general population may represent undiagnosed 22q11.2DS. Increased recognition of the syndrome by family doctors, specialists and coroners will be essential to facilitate the tissue studies needed to determine underlying mechanisms.

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Year:  2009        PMID: 19246480      PMCID: PMC3188306          DOI: 10.1136/jmg.2008.063800

Source DB:  PubMed          Journal:  J Med Genet        ISSN: 0022-2593            Impact factor:   6.318


  34 in total

1.  Frequency and predictive value of 22q11 deletion.

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Journal:  J Med Genet       Date:  1999-10       Impact factor: 6.318

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3.  Neuropathologic features in adults with 22q11.2 deletion syndrome.

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5.  Presenting phenotype in 100 children with the 22q11 deletion syndrome.

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10.  Phenotype of the 22q11.2 deletion in individuals identified through an affected relative: cast a wide FISHing net!

Authors:  D M McDonald-McGinn; M K Tonnesen; A Laufer-Cahana; B Finucane; D A Driscoll; B S Emanuel; E H Zackai
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1.  Caregiver and adult patient perspectives on the importance of a diagnosis of 22q11.2 deletion syndrome.

Authors:  G Costain; E W C Chow; P N Ray; A S Bassett
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Review 2.  Copy number variations in schizophrenia: critical review and new perspectives on concepts of genetics and disease.

Authors:  Anne S Bassett; Stephen W Scherer; Linda M Brzustowicz
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3.  Clinically detectable copy number variations in a Canadian catchment population of schizophrenia.

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4.  Pathogenic rare copy number variants in community-based schizophrenia suggest a potential role for clinical microarrays.

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5.  Practical guidelines for managing patients with 22q11.2 deletion syndrome.

Authors:  Anne S Bassett; Donna M McDonald-McGinn; Koen Devriendt; Maria Cristina Digilio; Paula Goldenberg; Alex Habel; Bruno Marino; Solveig Oskarsdottir; Nicole Philip; Kathleen Sullivan; Ann Swillen; Jacob Vorstman
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6.  Complex congenital heart disease in unaffected relatives of adults with 22q11.2 deletion syndrome.

Authors:  Jodi-Ann M Swaby; Candice K Silversides; Sean C Bekeschus; Sara Piran; Erwin N Oechslin; Eva W C Chow; Anne S Bassett
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Review 7.  Neuropsychiatric expression and catatonia in 22q11.2 deletion syndrome: An overview and case series.

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9.  Association between early-onset Parkinson disease and 22q11.2 deletion syndrome: identification of a novel genetic form of Parkinson disease and its clinical implications.

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10.  Undiagnosed DiGeorge syndrome presenting in middle age with an aortic root aneurysm and chronic dissection.

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