Literature DB >> 19219554

Myocardial adaptations in the failing heart: cause or consequence?

Sabine J van Dijk1, Nazha Hamdani, Ger J M Stienen, Jolanda van der Velden.   

Abstract

Many changes in morphology, biochemical properties and myocyte function occur during development to heart failure. Most changes may be compensatory, yet unable to prevent cardiac dysfunction in the long run. This illustrates that it is important to carefully dissect the disease causing modifications from cardiac adaptation, in order to obtain a better understanding of the pathophysiological processes leading to heart failure.

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Year:  2009        PMID： 19219554     DOI： 10.1007/s10974-009-9169-x

Source DB:  PubMed          Journal:  J Muscle Res Cell Motil        ISSN： 0142-4319            Impact factor:   2.698


  29 in total

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5.  Myosin heavy chain gene expression in human heart failure.

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6.  Regional remodeling strain and its association with myocardial apoptosis after myocardial infarction in an ovine model.

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Review 8.  Angiotensin receptor blockers: current status and future prospects.

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Review 2.  A piece of the human heart: variance of protein phosphorylation in left ventricular samples from end-stage primary cardiomyopathy patients.

Authors:  Sabine J van Dijk; Rozemarije A Holewijn; Anouk Tebeest; Cris Dos Remedios; Ger J M Stienen; Jolanda van der Velden
Journal:  J Muscle Res Cell Motil       Date:  2010-03-06       Impact factor: 2.698

Review 3.  Cardiac myosin binding protein C phosphorylation in cardiac disease.

Authors:  Diederik W D Kuster; Amira Cholid Bawazeer; Ruud Zaremba; Max Goebel; Nicky M Boontje; Jolanda van der Velden
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4.  FRET biosensor uncovers cAMP nano-domains at β-adrenergic targets that dictate precise tuning of cardiac contractility.

Authors:  Nicoletta C Surdo; Marco Berrera; Andreas Koschinski; Marcella Brescia; Matias R Machado; Carolyn Carr; Peter Wright; Julia Gorelik; Stefano Morotti; Eleonora Grandi; Donald M Bers; Sergio Pantano; Manuela Zaccolo
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  4 in total

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