Literature DB >> 19201342

Effect of etiology and timing of respiratory tract infections on development of bronchiolitis obliterans syndrome.

Vincent G Valentine1, Meera R Gupta, James E Walker, Leonardo Seoane, Ryan W Bonvillain, Gisele A Lombard, David Weill, Gundeep S Dhillon.   

Abstract

BACKGROUND: Among the many potential risk factors influencing the development of bronchiolitis obliterans syndrome (BOS), acute cellular rejection is the most frequently identified. Despite the unique susceptibility of the lung allograft to pathogens, the association with respiratory tract infections remains unclear. In this study we analyze the role respiratory tract infections have on the development of BOS after lung transplantation.
METHODS: Data from a single center were analyzed from 161 lung recipients transplanted from November 1990 to November 2005, and who survived >180 days. Univariate and multivariate Cox regression analyses were performed using BOS development and the time-scale was reported with hazard ratios (HRs) and confidence intervals (CIs).
RESULTS: Significant findings by univariate analysis per 100 patient-days prior to BOS onset included acute rejection, cytomegalovirus (CMV) pneumonitis, Gram-negative respiratory tract infections, Gram-positive respiratory tract infections and fungal pneumonias. Multivariate analysis indicated acute rejection, Gram-negative, Gram-positive and fungal pneumonias with HRs (CI) of 84 (23 to 309), 6.6 (1.2 to 37), 6,371 (84 to 485,000) and 314 (53 to 1,856) to be associated with BOS, respectively. Acute rejection, CMV pneumonitis, Gram-positive pneumonia and fungal pneumonitis in the first 100 days had HRs (CI) of 1.8 (1.1 to 3.2), 3.1 (1.3 to 6.9), 3.8 (1.5 to 9.4) and 2.1 (1.1 to 4.0), respectively, and acute rejection and fungal pneumonitis in the late post-operative period with HRs (CI) of 2.3 (1.2 to 4.4) and 1.5 (1.1 to 1.9), respectively.
CONCLUSIONS: In addition to acute rejection, pneumonias with GP, GN and fungal pathogens occurring prior to BOS are independent determinants of chronic allograft dysfunction. Early recognition and treatment of these pathogens in lung transplant recipients may improve long-term outcomes after transplantation.

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Year:  2009        PMID: 19201342     DOI: 10.1016/j.healun.2008.11.907

Source DB:  PubMed          Journal:  J Heart Lung Transplant        ISSN: 1053-2498            Impact factor:   10.247


  29 in total

1.  Telomere length in patients with pulmonary fibrosis associated with chronic lung allograft dysfunction and post-lung transplantation survival.

Authors:  Chad A Newton; Julia Kozlitina; Jefferson R Lines; Vaidehi Kaza; Fernando Torres; Christine Kim Garcia
Journal:  J Heart Lung Transplant       Date:  2017-02-04       Impact factor: 10.247

2.  Respiratory virus-induced dysregulation of T-regulatory cells leads to chronic rejection.

Authors:  Ankit Bharat; Elbert Kuo; Deepti Saini; Nancy Steward; Ramsey Hachem; Elbert P Trulock; G Alexander Patterson; Bryan F Meyers; Thalachallour Mohanakumar
Journal:  Ann Thorac Surg       Date:  2010-11       Impact factor: 4.330

3.  Insights from the European Respiratory Society 2018 Annual International Congress in the fields of thoracic surgery and lung transplantation.

Authors:  Rogier A S Hoek; Stelios Gaitanakis; Merel E Hellemons
Journal:  J Thorac Dis       Date:  2018-09       Impact factor: 2.895

Review 4.  Infections in the immunosuppressed host.

Authors:  M Patricia George; Henry Masur; Karen A Norris; Scott M Palmer; Cornelius J Clancy; John F McDyer
Journal:  Ann Am Thorac Soc       Date:  2014-08

5.  Non-tuberculous mycobacterium infection after lung transplantation is associated with increased mortality.

Authors:  Hsuanwen C Huang; S Samuel Weigt; Ariss Derhovanessian; Vyacheslav Palchevskiy; Abbas Ardehali; Rajan Saggar; Rajeev Saggar; Bernard Kubak; Aric Gregson; David J Ross; Joseph P Lynch; Robert Elashoff; John A Belperio
Journal:  J Heart Lung Transplant       Date:  2011-04-08       Impact factor: 10.247

6.  Staphylococcus via an interaction with the ELR+ CXC chemokine ENA-78 is associated with BOS.

Authors:  A L Gregson; X Wang; P Injean; S S Weigt; M Shino; D Sayah; A DerHovanessian; J P Lynch; D J Ross; R Saggar; A Ardehali; G Li; R Elashoff; J A Belperio
Journal:  Am J Transplant       Date:  2015-02-12       Impact factor: 8.086

7.  Mannose-binding lectin deficiency linked to cytomegalovirus (CMV) reactivation and survival in lung transplantation.

Authors:  J M Kwakkel-van Erp; A W M Paantjens; D A van Kessel; J C Grutters; J M M van den Bosch; E A van de Graaf; H G Otten
Journal:  Clin Exp Immunol       Date:  2011-06-27       Impact factor: 4.330

8.  Using mobile health technology to deliver decision support for self-monitoring after lung transplantation.

Authors:  Yun Jiang; Susan M Sereika; Annette DeVito Dabbs; Steven M Handler; Elizabeth A Schlenk
Journal:  Int J Med Inform       Date:  2016-07-19       Impact factor: 4.046

9.  Interaction between Pseudomonas and CXC chemokines increases risk of bronchiolitis obliterans syndrome and death in lung transplantation.

Authors:  Aric L Gregson; Xiaoyan Wang; S Sam Weigt; Vyacheslav Palchevskiy; Joseph P Lynch; David J Ross; Bernard M Kubak; Rajan Saggar; Michael C Fishbein; Abbas Ardehali; Gang Li; Robert Elashoff; John A Belperio
Journal:  Am J Respir Crit Care Med       Date:  2013-01-17       Impact factor: 21.405

10.  Graft Loss and CLAD-Onset Is Hastened by Viral Pneumonia After Lung Transplantation.

Authors:  Paul R Allyn; Erin L Duffy; Romney M Humphries; Patil Injean; S Samuel Weigt; Rajan Saggar; Michael Y Shino; Joseph P Lynch; Abbas Ardehali; Bernard Kubak; Chi-Hong Tseng; John A Belperio; David J Ross; Aric L Gregson
Journal:  Transplantation       Date:  2016-11       Impact factor: 4.939

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