Literature DB >> 19194191

A prospective appraisal of pulmonary hypertension in children with sickle cell disease.

Aziza Sedrak1, Sreedhar P Rao, Scott T Miller, Vahid Hekmat, Madu Rao.   

Abstract

OBJECTIVES: Pulmonary hypertension (PHT) is a life-threatening complication of sickle cell disease that occurs in 20% to 40% of adults. Measurement of maximal tricuspid regurgitant jet velocity (TRV) by echocardiography provides a noninvasive screening tool; TRV values > or =2.5 m/s are correlated with PHT and increased mortality. Our objective was to estimate the prevalence of PHT in our pediatric sickle cell population and its possible association with various clinical and laboratory findings, including obstructive sleep apnea and/or pulmonary dysfunction. STUDY
DESIGN: Eligible children had measurement of the TRV. Clinical data were collected, including detailed history with a standardized sleep apnea questionnaire; those with suggestive histories had polysomonography. Pulmonary function was assessed using whole body plethysmography.
RESULTS: Of 48 subjects (79% homozygous sickle cell anemia; median age 12 y; 11 receiving chronic transfusion) enrolled in the study, 4 (8.3%) had TRV >2.5 m/s; all had homozygous sickle cell anemia and 1 was receiving hydroxyurea after 3 years of transfusion for secondary stroke prevention. Subjects with elevated TRV had higher indirect bilirubin levels; we found no association between elevated TRV and obstructive apnea or pulmonary function abnormalities.
CONCLUSIONS: Elevation of TRV was relatively uncommon in our pediatric patients as compared with prevalence reported in adults and may be associated with increased hemolysis. There was no association with obstructive sleep apnea or abnormal pulmonary function.

Entities:  

Mesh:

Substances:

Year:  2009        PMID: 19194191     DOI: 10.1097/MPH.0b013e31818e5343

Source DB:  PubMed          Journal:  J Pediatr Hematol Oncol        ISSN: 1077-4114            Impact factor:   1.289


  6 in total

1.  Elevated tricuspid regurgitation velocity and decline in exercise capacity over 22 months of follow up in children and adolescents with sickle cell anemia.

Authors:  Victor R Gordeuk; Caterina P Minniti; Mehdi Nouraie; Andrew D Campbell; Sohail R Rana; Lori Luchtman-Jones; Craig Sable; Niti Dham; Gregory Ensing; Josef T Prchal; Gregory J Kato; Mark T Gladwin; Oswaldo L Castro
Journal:  Haematologica       Date:  2010-09-30       Impact factor: 9.941

Review 2.  Pleiotropic effects of intravascular haemolysis on vascular homeostasis.

Authors:  Gregory J Kato; James G Taylor
Journal:  Br J Haematol       Date:  2009-12-01       Impact factor: 6.998

3.  Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directions.

Authors:  Gregory J Kato; Robert P Hebbel; Martin H Steinberg; Mark T Gladwin
Journal:  Am J Hematol       Date:  2009-09       Impact factor: 10.047

4.  Respiratory muscle force and lung volume changes in a population of children with sickle cell disease.

Authors:  Bruce A Ong; Jason Caboot; Abbas Jawad; Joseph McDonough; Tannoa Jackson; Raanan Arens; Carole L Marcus; Kim Smith-Whitley; Thornton B A Mason; Kwaku Ohene-Frempong; Julian L Allen
Journal:  Br J Haematol       Date:  2013-07-20       Impact factor: 6.998

5.  Doppler echocardiographic assessment of pulmonary artery pressure in children with sickle cell anaemia.

Authors:  Moriam Omolola Lamina; Barakat Adeola Animasahun; Ijeoma Nnena Akinwumi; Olisamedua Fidelis Njokanma
Journal:  Cardiovasc Diagn Ther       Date:  2019-06

6.  Low hemoglobin increases risk for cerebrovascular disease, kidney disease, pulmonary vasculopathy, and mortality in sickle cell disease: A systematic literature review and meta-analysis.

Authors:  Kenneth I Ataga; Victor R Gordeuk; Irene Agodoa; Jennifer A Colby; Kimberly Gittings; Isabel E Allen
Journal:  PLoS One       Date:  2020-04-03       Impact factor: 3.240

  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.