Literature DB >> 19160029

Polymorphisms of PTPN11 coding SHP-2 as biomarkers for ulcerative colitis susceptibility in the Japanese population.

Yukiko Narumi1, Hajime Isomoto, Mizuho Shiota, Kayoko Sato, Shinji Kondo, Haruhisa Machida, Katsunori Yanagihara, Yohei Mizuta, Shigeru Kohno, Kazuhiro Tsukamoto.   

Abstract

OBJECTIVE: To identify genetic determinants of inflammatory bowel disease (IBD), we examined an association between polymorphisms of both the programmed cell death 1 gene (PDCD1) and the src homology 2 domain-containing tyrosine phosphatase 2 gene (PTPN11) and susceptibility to IBD.
METHODS: Study subjects comprised 114 patients with ulcerative colitis (UC), 83 patients with Crohn's disease, and 200 healthy control subjects. Five single nucleotide polymorphisms (SNPs) in PDCD1 and PTPN11 were detected by polymerase chain reaction restriction fragment length polymorphism. Subsequently, haplotypes composed of the two SNPs in PTPN11 were constructed.
RESULTS: The frequencies of the Hap 1 haplotype and its homozygous Hap 1/Hap 1 diplotype of PTPN11 were significantly increased in UC patients compared to control subjects (P = 0.011 and P = 0.030, respectively). While no association was found for PDCD1 for UC or CD and none for PTPN11 for CD.
CONCLUSION: PTPN11 is a genetic determinant for the pathogenesis of UC, and haplotyping of PTPN11 may be useful as a genetic biomarker to identify high-risk individuals susceptible to UC.

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Year:  2009        PMID: 19160029     DOI: 10.1007/s10875-008-9272-6

Source DB:  PubMed          Journal:  J Clin Immunol        ISSN: 0271-9142            Impact factor:   8.317


  44 in total

1.  Tyrosine phosphatase SHP-2 binding to CTLA-4: absence of direct YVKM/YFIP motif recognition.

Authors:  H Schneider; C E Rudd
Journal:  Biochem Biophys Res Commun       Date:  2000-03-05       Impact factor: 3.575

2.  PD-1 immunoreceptor inhibits B cell receptor-mediated signaling by recruiting src homology 2-domain-containing tyrosine phosphatase 2 to phosphotyrosine.

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3.  Correlation between PTPN11 gene mutations and congenital heart defects in Noonan and LEOPARD syndromes.

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5.  Association of polymorphic alleles of CTLA4 with inflammatory bowel disease in the Japanese.

Authors:  Haruhisa Machida; Kazuhiro Tsukamoto; Chun-Yang Wen; Yukiko Narumi; Saburou Shikuwa; Hajime Isomoto; Fuminao Takeshima; Yohei Mizuta; Norio Niikawa; Ikuo Murata; Shigeru Kohno
Journal:  World J Gastroenterol       Date:  2005-07-21       Impact factor: 5.742

6.  Somatic mutations in PTPN11 in juvenile myelomonocytic leukemia, myelodysplastic syndromes and acute myeloid leukemia.

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10.  Grouping of multiple-lentigines/LEOPARD and Noonan syndromes on the PTPN11 gene.

Authors:  Maria Cristina Digilio; Emanuela Conti; Anna Sarkozy; Rita Mingarelli; Tania Dottorini; Bruno Marino; Antonio Pizzuti; Bruno Dallapiccola
Journal:  Am J Hum Genet       Date:  2002-06-07       Impact factor: 11.025

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  17 in total

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Review 6.  New and Unexpected Biological Functions for the Src-Homology 2 Domain-Containing Phosphatase SHP-2 in the Gastrointestinal Tract.

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7.  SHP-2 Phosphatase Prevents Colonic Inflammation by Controlling Secretory Cell Differentiation and Maintaining Host-Microbiota Homeostasis.

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8.  SHP-2 phosphatase contributes to KRAS-driven intestinal oncogenesis but prevents colitis-associated cancer development.

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9.  SHP2 associates with nuclear localization of STAT3: significance in progression and prognosis of colorectal cancer.

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10.  Association between single-nucleotide polymorphisms and adverse events in nivolumab-treated non-small cell lung cancer patients.

Authors:  Sander Bins; Edwin A Basak; Samira El Bouazzaoui; Stijn L W Koolen; E Oomen-de Hoop; Cor H van der Leest; Astrid A M van der Veldt; Stefan Sleijfer; Reno Debets; Ron H N van Schaik; Joachim G J V Aerts; Ron H J Mathijssen
Journal:  Br J Cancer       Date:  2018-04-26       Impact factor: 7.640

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