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8 in total

1. Basophil CD203c levels are increased at baseline and can be used to monitor omalizumab treatment in subjects with nut allergy.

Authors: Yael Gernez; Rabindra Tirouvanziam; Grace Yu; Eliver E B Ghosn; Neha Reshamwala; Tammie Nguyen; Mindy Tsai; Stephen J Galli; Leonard A Herzenberg; Leonore A Herzenberg; Kari C Nadeau
Journal: Int Arch Allergy Immunol Date: 2010-10-25 Impact factor: 2.749

2. Immuno-evasive tactics by schistosomes identify an effective allergy preventative.

Authors: Qyana Griffith; YanMei Liang; Patrick Whitworth; Carlos Rodriguez-Russo; Ahmad Gul; Afzal A Siddiqui; John Connor; Pauline Mwinzi; Lisa Ganley-Leal
Journal: Exp Parasitol Date: 2015-03-24 Impact factor: 2.011

3. Effective treatment of different phenotypes of chronic urticaria with omalizumab: Case reports and review of literature.

Authors: A Kasperska-Zajac; J Jarząb; A Żerdzińska; K Bąk; A Grzanka
Journal: Int J Immunopathol Pharmacol Date: 2016-01-04 Impact factor: 3.219

Review 8. Regulation of Trafficking and Signaling of the High Affinity IgE Receptor by FcεRIβ and the Potential Impact of FcεRIβ Splicing in Allergic Inflammation.

Authors: Greer K Arthur; Glenn Cruse
Journal: Int J Mol Sci Date: 2022-01-12 Impact factor: 5.923

8 in total

Therapeutic efficacy of omalizumab.

1. Basophil CD203c levels are increased at baseline and can be used to monitor omalizumab treatment in subjects with nut allergy.

2. Immuno-evasive tactics by schistosomes identify an effective allergy preventative.

3. Effective treatment of different phenotypes of chronic urticaria with omalizumab: Case reports and review of literature.

4. Effects of omalizumab on basophil and mast cell responses using an intranasal cat allergen challenge.

Review 5. IgE and mast cells in allergic disease.

6. Cytomegalic hepatitis in a patient receiving omalizumab.

7. Antibody to FcεRIα Suppresses Immunoglobulin E Binding to High-Affinity Receptor I in Allergic Inflammation.

Review 8. Regulation of Trafficking and Signaling of the High Affinity IgE Receptor by FcεRIβ and the Potential Impact of FcεRIβ Splicing in Allergic Inflammation.