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Literature DB >> 19095955

The effects of AGEing on diet.

Timo Buetler, Thomas Henle.

Abstract

Mesh：

Substances：

Year: 2008 PMID： 19095955 PMCID： PMC2631347 DOI： 10.2353/ajpath.2009.080805

Source DB: PubMed Journal: Am J Pathol ISSN： 0002-9440 Impact factor: 4.307

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16 in total

1. The effects of partial thiamin deficiency and oxidative stress (i.e., glyoxal and methylglyoxal) on the levels of alpha-oxoaldehyde plasma protein adducts in Fischer 344 rats.

Authors: Nandita Shangari; Flore Depeint; Rudolf Furrer; W Robert Bruce; Peter J O'Brien
Journal: FEBS Lett Date: 2005-09-28 Impact factor: 4.124

2. Inflammatory mediators are induced by dietary glycotoxins, a major risk factor for diabetic angiopathy.

Authors: Helen Vlassara; Weijing Cai; Jill Crandall; Teresia Goldberg; Robert Oberstein; Veronique Dardaine; Melpomeni Peppa; Elliot J Rayfield
Journal: Proc Natl Acad Sci U S A Date: 2002-11-12 Impact factor: 11.205

3. Restriction of dietary glycotoxins reduces excessive advanced glycation end products in renal failure patients.

Authors: Jaime Uribarri; Melpomeni Peppa; Weijing Cai; Teresia Goldberg; Min Lu; Cijiang He; Helen Vlassara
Journal: J Am Soc Nephrol Date: 2003-03 Impact factor: 10.121

4. Effects of a diet rich in advanced glycation end products in the rat remnant kidney model.

Authors: Katarína Sebeková; Veronika Faist; Thomas Hofmann; Reinhard Schinzel; August Heidland
Journal: Am J Kidney Dis Date: 2003-03 Impact factor: 8.860

5. High levels of dietary advanced glycation end products transform low-density lipoprotein into a potent redox-sensitive mitogen-activated protein kinase stimulant in diabetic patients.

Authors: Weijing Cai; John Cijiang He; Li Zhu; Melpomeni Peppa; Changyong Lu; Jaime Uribarri; Helen Vlassara
Journal: Circulation Date: 2004-07-12 Impact factor: 29.690

6. AGE/RAGE produces endothelial dysfunction in coronary arterioles in type 2 diabetic mice.

Authors: Xue Gao; Hanrui Zhang; Ann Marie Schmidt; Cuihua Zhang
Journal: Am J Physiol Heart Circ Physiol Date: 2008-06-06 Impact factor: 4.733

7. Lowering of dietary advanced glycation endproducts (AGE) reduces neointimal formation after arterial injury in genetically hypercholesterolemic mice.

Authors: Reigh-Yi Lin; Ernane D Reis; Anthony T Dore; Min Lu; Newsha Ghodsi; John T Fallon; Edward A Fisher; Helen Vlassara
Journal: Atherosclerosis Date: 2002-08 Impact factor: 5.162

Review 8. Advanced glycation end products and vascular inflammation: implications for accelerated atherosclerosis in diabetes.

Authors: Giuseppina Basta; Ann Marie Schmidt; Raffaele De Caterina
Journal: Cardiovasc Res Date: 2004-09-01 Impact factor: 10.787

The effects of AGEing on diet.

1. The effects of partial thiamin deficiency and oxidative stress (i.e., glyoxal and methylglyoxal) on the levels of alpha-oxoaldehyde plasma protein adducts in Fischer 344 rats.

2. Inflammatory mediators are induced by dietary glycotoxins, a major risk factor for diabetic angiopathy.

3. Restriction of dietary glycotoxins reduces excessive advanced glycation end products in renal failure patients.

4. Effects of a diet rich in advanced glycation end products in the rat remnant kidney model.

5. High levels of dietary advanced glycation end products transform low-density lipoprotein into a potent redox-sensitive mitogen-activated protein kinase stimulant in diabetic patients.

6. AGE/RAGE produces endothelial dysfunction in coronary arterioles in type 2 diabetic mice.

7. Lowering of dietary advanced glycation endproducts (AGE) reduces neointimal formation after arterial injury in genetically hypercholesterolemic mice.

Review 8. Advanced glycation end products and vascular inflammation: implications for accelerated atherosclerosis in diabetes.

9. Oxidative stress-inducing carbonyl compounds from common foods: novel mediators of cellular dysfunction.

10. Dietary glycotoxins correlate with circulating advanced glycation end product levels in renal failure patients.

Review 1. Dietary Advanced Glycation Endproducts and the Gastrointestinal Tract.

2. Advanced Glycation End Products Induce Obesity and Hepatosteatosis in CD-1 Wild-Type Mice.