Literature DB >> 19091790

Role of CuZn superoxide dismutase on carotid body function in heart failure rabbits.

Yanfeng Ding1, Yu-Long Li, Matthew C Zimmerman, Robin L Davisson, Harold D Schultz.   

Abstract

AIMS: Peripheral chemoreflex sensitivity is potentiated in both clinical and experimental chronic heart failure (CHF). NADPH oxidase-derived superoxide mediates angiotensin II (Ang II)-enhanced carotid body (CB) chemoreceptor sensitivity in CHF rabbits, and tempol, the superoxide dismutase (SOD) mimetic, inhibits this Ang II- and CHF-enhanced superoxide anion effect. Here we investigated the role of cytoplasmic SOD [CuZn superoxide dismutase (CuZnSOD)] in the CB on chemoreceptor activity and function in CHF rabbits. METHODS AND
RESULTS: CuZnSOD protein expression was decreased in CBs from CHF rabbits vs. sham (P < 0.05). Adenoviral CuZnSOD (Ad CuZnSOD) gene transfer to the CBs increased CuZnSOD protein expression and significantly reduced the baseline renal sympathetic nerve activity (RSNA) and the response of RSNA to hypoxia in the CHF rabbits (P < 0.05). Single-fibre discharge from CB chemoafferents during normoxia (baseline, at approximately 100 mmHg PO2) and in response to hypoxia were enhanced in CHF vs. sham rabbits (P < 0.05). Ad CuZnSOD decreased the baseline discharge (7.6 +/- 1.3 vs. 12.6 +/- 1.7 imp/s at approximately 100 mmHg PO2) and the response to hypoxia (22.4 +/- 1.6 vs. 32.3 +/- 1.2 imp/s at approximately 40 mmHg PO2, P < 0.05) in CHF rabbits. Ad CuZnSOD also normalized the blunted outward K+ current (IK) in CB glomus cells from CHF rabbits (369 +/- 14 vs. 565 +/- 31 pA/pF at +70 mV, P < 0.05). In addition, Ad CuZnSOD reduced the elevation of superoxide level in CBs from CHF rabbits.
CONCLUSION: Downregulation of CuZnSOD in the CB contributes to the enhanced activity of CB chemoreceptors and chemoreflex function in CHF rabbits.

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Year:  2008        PMID: 19091790      PMCID: PMC2642603          DOI: 10.1093/cvr/cvn350

Source DB:  PubMed          Journal:  Cardiovasc Res        ISSN: 0008-6363            Impact factor:   10.787


  29 in total

1.  Gene transfer of neuronal nitric oxide synthase to carotid body reverses enhanced chemoreceptor function in heart failure rabbits.

Authors:  Yu-Long Li; Yi-Fan Li; Dongmei Liu; Kurtis G Cornish; Kaushik P Patel; Irving H Zucker; Keith M Channon; Harold D Schultz
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2.  Enhanced peripheral chemoreflex function in conscious rabbits with pacing-induced heart failure.

Authors:  S Y Sun; W Wang; I H Zucker; H D Schultz
Journal:  J Appl Physiol (1985)       Date:  1999-04

3.  Enhanced activity of carotid body chemoreceptors in rabbits with heart failure: role of nitric oxide.

Authors:  S Y Sun; W Wang; I H Zucker; H D Schultz
Journal:  J Appl Physiol (1985)       Date:  1999-04

4.  Augmented peripheral chemosensitivity as a potential input to baroreflex impairment and autonomic imbalance in chronic heart failure.

Authors:  P Ponikowski; T P Chua; M Piepoli; D Ondusova; K Webb-Peploe; D Harrington; S D Anker; M Volterrani; R Colombo; G Mazzuero; A Giordano; A J Coats
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Review 6.  Chemoreflex function in heart failure.

Authors:  H D Schultz; S Y Sun
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7.  Clinical characteristics of chronic heart failure patients with an augmented peripheral chemoreflex.

Authors:  T P Chua; P Ponikowski; K Webb-Peploe; D Harrington; S D Anker; M Piepoli; A J Coats
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8.  Ventilatory response to exercise correlates with impaired heart rate variability in patients with chronic congestive heart failure.

Authors:  P Ponikowski; T P Chua; M Piepoli; W Banasiak; S D Anker; R Szelemej; W Molenda; K Wrabec; A Capucci; A J Coats
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Review 9.  Neural regulation of sympathetic nerve activity in heart failure.

Authors:  I H Zucker; W Wang; M Brändle; H D Schultz; K P Patel
Journal:  Prog Cardiovasc Dis       Date:  1995 May-Jun       Impact factor: 8.194

Review 10.  Sympathetic dysregulation in heart failure: mechanisms and therapy.

Authors:  A L Mark
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  37 in total

1.  Role of blood flow in carotid body chemoreflex function in heart failure.

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2.  SOD1 overexpression in paraventricular nucleus improves post-infarct myocardial remodeling and ventricular function.

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6.  Neuronal uptake of nanoformulated superoxide dismutase and attenuation of angiotensin II-dependent hypertension after central administration.

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7.  Ventilatory responses to chemoreflex stimulation are not enhanced by angiotensin II in healthy humans.

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Review 8.  Angiotensin II, sympathetic nerve activity and chronic heart failure.

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Review 9.  Translating carotid body function into clinical medicine.

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10.  Cardiac diastolic and autonomic dysfunction are aggravated by central chemoreflex activation in heart failure with preserved ejection fraction rats.

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