Literature DB >> 19083041

Effects of high-AGE beverage on RAGE and VEGF expressions in the liver and kidneys.

Takashi Sato1, Xuegang Wu, Noriko Shimogaito, Jun-ichi Takino, Sho-ichi Yamagishi, Masayoshi Takeuchi.   

Abstract

BACKGROUND: The formation and accumulation of advanced glycation end products (AGEs) increase in some lifestyle-related diseases as well as in aging; however, little is known about the relationship between food-derived AGEs and the pathology of such diseases. AIM OF THE STUDY AND METHODS: To explore whether food items containing high levels of AGEs are involved in the development of lifestyle-related diseases, rats were orally administered a commercial high-AGE beverage [Lactobacillus beverage-A (LB-A)]. With a particular focus on angiogenesis-associated diseases, the gene expressions of vascular endothelial growth factor (VEGF) and the receptor for AGEs (RAGE) were examined in the liver and kidneys using real-time reverse transcription-polymerase chain reaction. Moreover, AGE deposition was immunohistochemically investigated in these tissues. RESULTS AND
CONCLUSIONS: Hepatic VEGF expression was significantly increased in rats administered LB-A (P < 0.01 vs. control). Furthermore, immunohistochemical analysis detected glucose-derived AGE-positive cells in the liver from the LB-A group. These results suggest that AGE-rich beverages increase hepatic VEGF expression and AGE accumulation, bringing about early events associated with lifestyle-related diseases.

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Year:  2008        PMID: 19083041     DOI: 10.1007/s00394-008-0753-4

Source DB:  PubMed          Journal:  Eur J Nutr        ISSN: 1436-6207            Impact factor:   5.614


  26 in total

1.  Advanced glycation end products are eliminated by scavenger-receptor-mediated endocytosis in hepatic sinusoidal Kupffer and endothelial cells.

Authors:  B Smedsrød; J Melkko; N Araki; H Sano; S Horiuchi
Journal:  Biochem J       Date:  1997-03-01       Impact factor: 3.857

2.  Regulation of vascular endothelial growth factor expression by advanced glycation end products.

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3.  Inflammatory mediators are induced by dietary glycotoxins, a major risk factor for diabetic angiopathy.

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4.  Detection of noncarboxymethyllysine and carboxymethyllysine advanced glycation end products (AGE) in serum of diabetic patients.

Authors:  M Takeuchi; Z Makita; K Yanagisawa; Y Kameda; T Koike
Journal:  Mol Med       Date:  1999-06       Impact factor: 6.354

Review 5.  Advanced glycation endproducts--role in pathology of diabetic complications.

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10.  Lowering of dietary advanced glycation endproducts (AGE) reduces neointimal formation after arterial injury in genetically hypercholesterolemic mice.

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  11 in total

1.  Advanced glycation end products promote differentiation of CD4(+) T helper cells toward pro-inflammatory response.

Authors:  Xiao-Qun Han; Zuo-Jiong Gong; San-Qing Xu; Xun Li; Li-Kun Wang; Shi-Min Wu; Jian-Hong Wu; Hua-Fen Yang
Journal:  J Huazhong Univ Sci Technolog Med Sci       Date:  2014-02-06

2.  Natural history of age-related retinal lesions that precede AMD in mice fed high or low glycemic index diets.

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Review 3.  Involvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategies.

Authors:  Masayoshi Takeuchi; Jun-Ichi Takino; Akiko Sakasai-Sakai; Takanobu Takata; Tadashi Ueda; Mikihiro Tsutsumi; Hideyuki Hyogo; Sho-Ichi Yamagishi
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4.  Long-term treatment with spermidine increases health span of middle-aged Sprague-Dawley male rats.

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Review 5.  Role of receptor for advanced glycation end products (RAGE) in liver disease.

Authors:  Sho-ichi Yamagishi; Takanori Matsui
Journal:  Eur J Med Res       Date:  2015-02-11       Impact factor: 2.175

6.  Assessment of the concentrations of various advanced glycation end-products in beverages and foods that are commonly consumed in Japan.

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Review 8.  Current perspectives on the health risks associated with the consumption of advanced glycation end products: recommendations for dietary management.

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9.  Synchrotron infrared imaging of advanced glycation endproducts (AGEs) in cardiac tissue from mice fed high glycemic diets.

Authors:  Giovanni Birarda; Elizabeth A Holman; Shang Fu; Karen Weikel; Ping Hu; Francis G Blankenberg; Hoi-Ying Holman; Allen Taylor
Journal:  Biomed Spectrosc Imaging       Date:  2013

Review 10.  Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases.

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Journal:  Diagnostics (Basel)       Date:  2016-06-07
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