Literature DB >> 19034449

Stable XIAP knockdown clones of HCT116 colon cancer cells are more sensitive to TRAIL, taxanes and irradiation in vitro.

Kate Connolly1, Richard Mitter, Morwenna Muir, Duncan Jodrell, Sylvie Guichard.   

Abstract

PURPOSE: To develop a model of X-linked inhibitor of apoptosis (XIAP) down regulation in colorectal cancer cell lines. This may be used to determine whether combination strategies have clinical potential.
METHODS: A series of clones were developed using short hairpin RNA (shRNA) against XIAP stably expressed in HCT116 cells. XIAP mRNA and protein levels were established by RT-PCR and Immunoblot, respectively. GeneChip microarrays confirmed XIAP knockdown and absence of compensation by other IAP members.
RESULTS: Four XIAP knockdown cell lines show 82-93% reduction in XIAP mRNA and 67-89% reduction in protein when compared to four luciferase control cell lines. XIAP knockdown sensitises cells to rhTRAIL by a factor of 3, to paclitaxel and docetaxel by a factor of >2 and, to a lesser extent, radiotherapy (20% enhancement).
CONCLUSIONS: Clinical trials with XIAP antisense continue, and these data suggest combination studies with agents such as rhTRAIL and taxanes should be undertaken.

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Year:  2008        PMID: 19034449     DOI: 10.1007/s00280-008-0872-x

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  11 in total

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10.  Prognostic significance of XIAP and NF-κB expression in esophageal carcinoma with postoperative radiotherapy.

Authors:  Suna Zhou; Wenguang Ye; Qiuju Shao; Yuhong Qi; Mingxin Zhang; Jun Liang
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