Literature DB >> 19034397

Control of allograft rejection in mice by applying a novel neuropeptide, cortistatin.

Jiang Wang1, Rong Zhao, Fuqin Zhang, Jianping Li, Binliang Huo, Yunxin Cao, Kefeng Dou.   

Abstract

INTRODUCTION: The action of cortistatin (CST), a novel cyclic neuropeptide, as an anti-inflammatory factor has been studied, but few investigations have explored the immunomodulatory role of CST in transplantation. In the present study, we examined whether CST affects the alloimmune response in a mouse model of skin transplantation and the effects of CST on T lymphocytes.
METHODS: BALB/c (H-2K(d)) recipient mice (n=70) were divided into seven groups (n=10 per group) and given an intraperitoneal injection of CST or a somatostatin analog, SMS 201-995 (octreotide), on the day of skin transplantation from C57BL/6 (B6) (H-2K(b)) donors. Injections were continued for 7 consecutive days. Groups 1-3 received CST at doses of 0.02, 0.2, or 2 mg/kg, respectively. Groups 4-6 received SMS 201-995 at the same doses. Group 7 was a control group and received injections of phosphate buffered saline. Survival of the allografts was recorded. A semiquantitative reverse transcriptase polymerase chain reaction study of Foxp3 expression and a flow cytometry study of CD4 and CD25 markers of T lymphocytes were conducted to determine whether CD4(+)CD25(+) Foxp3(high) regulatory T cells (T(reg)) were generated in vivo.
RESULTS: BALB/c mice given CST (0.2 or 2 mg/kg) had prolonged graft survival (median survival time [MST], 13 and 14 days, respectively; P<0.05 compared with controls). SMS 201-995 at the same concentrations did not have a significant effect on allograft survival (MST, 8 days for both groups). We found more than a twofold increase of CD4(+)CD25(+) T(reg) cells in the CD4(+) T-cell population and the expression of Foxp3 was up-regulated in the CST treatment groups, compared with control and SMS 201-995 treatment groups.
CONCLUSION: In our study, CST induced a significant prolongation in survival time of allogeneic skin grafts and increased the generation of CD4(+)CD25(+) Foxp 3(high) T(reg) cells. These results suggest that CST may become a new modality in controlling allograft rejection.

Entities:  

Mesh:

Substances:

Year:  2008        PMID: 19034397     DOI: 10.1007/s12325-008-0121-z

Source DB:  PubMed          Journal:  Adv Ther        ISSN: 0741-238X            Impact factor:   3.845


  4 in total

1.  The neuropeptide cortistatin attenuates experimental autoimmune myocarditis via inhibition of cardiomyogenic T cell-driven inflammatory responses.

Authors:  Virginia Delgado-Maroto; Clara P Falo; Irene Forte-Lago; Norma Adan; Maria Morell; Elena Maganto-Garcia; Gema Robledo; Francisco O'Valle; Andrew H Lichtman; Elena Gonzalez-Rey; Mario Delgado
Journal:  Br J Pharmacol       Date:  2017-01-08       Impact factor: 8.739

2.  DNA methylation is associated with inhaled corticosteroid response in persistent childhood asthmatics.

Authors:  Alberta L Wang; Olena Gruzieva; Weiliang Qiu; Simon Kebede Merid; Juan C Celedón; Benjamin A Raby; Cilla Söderhäll; Dawn L DeMeo; Scott T Weiss; Erik Melén; Kelan G Tantisira
Journal:  Clin Exp Allergy       Date:  2019-08-15       Impact factor: 5.018

3.  Decreased Expression of FOXP3 in Nasal Polyposis.

Authors:  Kannika Roongrotwattanasiri; Ruby Pawankar; Satoko Kimura; Sachiko Mori; Manabu Nonaka; Toshiaki Yagi
Journal:  Allergy Asthma Immunol Res       Date:  2011-11-09       Impact factor: 5.764

4.  The relationship between allergic status and adenotonsillar regrowth: a retrospective research on children after adenotonsillectomy.

Authors:  Zirong Huo; Jun Shi; Yilai Shu; Mingliang Xiang; Jingrong Lu; Hao Wu
Journal:  Sci Rep       Date:  2017-04-18       Impact factor: 4.379

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.