Literature DB >> 19033369

Suppression of gonadotropins and estradiol in premenopausal women by oral administration of the nonpeptide gonadotropin-releasing hormone antagonist elagolix.

R Scott Struthers1, Andrew J Nicholls, John Grundy, Takung Chen, Roland Jimenez, Samuel S C Yen, Haig P Bozigian.   

Abstract

CONTEXT: Parenteral administration of peptide GnRH analogs is widely employed for treatment of endometriosis and fibroids and in assisted-reproductive therapy protocols. Elagolix is a novel, orally available nonpeptide GnRH antagonist.
OBJECTIVE: Our objective was to evaluate the safety, pharmacokinetics, and inhibitory effects on gonadotropins and estradiol of single-dose and 7-d elagolix administration to healthy premenopausal women.
DESIGN: This was a first-in-human, double-blind, placebo-controlled, single- and multiple-dose study with sequential dose escalation. PARTICIPANTS: Fifty-five healthy, regularly cycling premenopausal women participated.
INTERVENTIONS: Subjects were administered a single oral dose of 25-400 mg or placebo. In a second arm of the study, subjects received placebo or 50, 100, or 200 mg once daily or 100 mg twice daily for 7 d. Treatment was initiated on d 7 (+/-1) after onset of menses. MAIN OUTCOME MEASURES: Safety, tolerability, pharmacokinetics, and serum LH, FSH, and estradiol concentrations were assessed.
RESULTS: Elagolix was well tolerated and rapidly bioavailable after oral administration. Serum gonadotropins declined rapidly. Estradiol was suppressed by 24 h in subjects receiving at least 50 mg/d. Daily (50-200 mg) or twice-daily (100 mg) administration for 7 d maintained low estradiol levels (17 +/- 3 to 68 +/- 46 pg/ml) in most subjects during late follicular phase. Effects of the compound were rapidly reversed after discontinuation.
CONCLUSIONS: Oral administration of a nonpeptide GnRH antagonist, elagolix, suppressed the reproductive endocrine axis in healthy premenopausal women. These results suggest that elagolix may enable dose-related pituitary and gonadal suppression in premenopausal women as part of treatment strategies for reproductive hormone-dependent disease states.

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Year:  2008        PMID: 19033369      PMCID: PMC2646513          DOI: 10.1210/jc.2008-1695

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  26 in total

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5.  Disappearance rates of endogenous luteinizing hormone and chorionic gonadotropin in man.

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6.  The dynamics of gonadotropin inhibition in women induced by an antagonistic analog of gonadotropin-releasing hormone.

Authors:  N S Cetel; J Rivier; W Vale; S S Yen
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9.  Reversible hypogonadism induced by a luteinizing hormone-releasing hormone (LH-RH) agonist (Buserelin) as a new therapeutic approach for endometriosis.

Authors:  A Lemay; R Maheux; N Faure; C Jean; A T Fazekas
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10.  Induction of hot flashes in premenopausal women treated with a long-acting GnRH agonist.

Authors:  J DeFazio; D R Meldrum; L Laufer; W Vale; J Rivier; J K Lu; H L Judd
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7.  Elagolix Alone or With Add-Back Therapy in Women With Heavy Menstrual Bleeding and Uterine Leiomyomas: A Randomized Controlled Trial.

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Review 9.  An Evidence-Based Review of Elagolix for the Treatment of Pain Secondary to Endometriosis.

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Review 10.  Elagolix in the treatment of heavy menstrual bleeding associated with uterine fibroids in premenopausal women.

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