Literature DB >> 19031041

Cerebral microdialysis of interleukin (IL)-1beta and IL-6: extraction efficiency and production in the acute phase after severe traumatic brain injury in rats.

Hedy Folkersma1, John J P Brevé, Fred J H Tilders, Leela Cherian, Claudia S Robertson, W Peter Vandertop.   

Abstract

BACKGROUND: As a research tool, cerebral microdialysis might be a useful technique in monitoring the release of cytokines into the extracellular fluid (ECF) following traumatic brain injury (TBI). We established extraction efficiency of Interleukin(IL)-1ss and Interleukin(IL)-6 by an in vitro microdialysis-perfusion system, followed by in vivo determination of the temporal profile of extracellular fluid cytokines after severe TBI in rats.
MATERIALS AND METHODS: In vitro experiments using a polyether sulfon (PES) microdialysis probe especially developed for recovery of macromolecules such as cytokines, were carried out to establish the extraction efficiency of IL-1ss and IL-6 from artificial cerebrospinal fluid (CSF) with defined IL-1ss and IL-6 concentrations. In vivo experiments in which rats were subjected to TBI or sham and microdialysis samples were collected from the parietal lobe for measurement of cytokines.
FINDINGS: The extraction efficiency was maximal 6.05% (range, 5.97-6.13%) at 0.5 microl/min(-1) and decreased at higher flow rates. Both cytokines were detectable in the dialysates. Highest IL-1ss levels were found within 200 min, highest IL-6 concentrations were detected at later intervals (200-400 min). No differences were found between the TBI and control groups.
CONCLUSIONS: Cerebral microdialysis allows measurement of cytokine secretion in the ECF of brain tissue in rats.

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Year:  2008        PMID: 19031041     DOI: 10.1007/s00701-008-0151-y

Source DB:  PubMed          Journal:  Acta Neurochir (Wien)        ISSN: 0001-6268            Impact factor:   2.216


  13 in total

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2.  The cytokine response to human traumatic brain injury: temporal profiles and evidence for cerebral parenchymal production.

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7.  Increased cerebral (R)-[(11)C]PK11195 uptake and glutamate release in a rat model of traumatic brain injury: a longitudinal pilot study.

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Review 9.  Endogenous repair signaling after brain injury and complementary bioengineering approaches to enhance neural regeneration.

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10.  Cytokine and Chemokine Recovery Is Increased by Colloid Perfusates during Dermal Microdialysis.

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