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19 in total

1. Hypoglossal motoneurons are postsynaptically inhibited during carbachol-induced rapid eye movement sleep.

Authors: J Yamuy; S J Fung; M Xi; F R Morales; M H Chase
Journal: Neuroscience Date: 1999 Impact factor: 3.590

2. Noradrenergic, serotonergic and GABAergic antagonists injected together into the XII nucleus abolish the REM sleep-like depression of hypoglossal motoneuronal activity.

Authors: Victor B Fenik; Richard O Davies; Leszek Kubin
Journal: J Sleep Res Date: 2005-12 Impact factor: 3.981

3. REM sleep-like atonia of hypoglossal (XII) motoneurons is caused by loss of noradrenergic and serotonergic inputs.

Authors: Victor B Fenik; Richard O Davies; Leszek Kubin
Journal: Am J Respir Crit Care Med Date: 2005-08-11 Impact factor: 21.405

4. Disinhibition of perifornical hypothalamic neurones activates noradrenergic neurones and blocks pontine carbachol-induced REM sleep-like episodes in rats.

Authors: Jackie W Lu; Victor B Fenik; Jennifer L Branconi; Graziella L Mann; Irma Rukhadze; Leszek Kubin
Journal: J Physiol Date: 2007-05-10 Impact factor: 5.182

5. The postsynaptic inhibitory control of lumbar motoneurons during the atonia of active sleep: effect of strychnine on motoneuron properties.

Authors: P J Soja; F López-Rodríguez; F R Morales; M H Chase
Journal: J Neurosci Date: 1991-09 Impact factor: 6.167

6. Evidence that glycine mediates the postsynaptic potentials that inhibit lumbar motoneurons during the atonia of active sleep.

Authors: M H Chase; P J Soja; F R Morales
Journal: J Neurosci Date: 1989-03 Impact factor: 6.167

7. Behavioral state-specific inhibitory postsynaptic potentials impinge on cat lumbar motoneurons during active sleep.

Authors: F R Morales; P Boxer; M H Chase
Journal: Exp Neurol Date: 1987-11 Impact factor: 5.330

8. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia.

Authors: Patricia L Brooks; John H Peever
Journal: J Neurosci Date: 2008-04-02 Impact factor: 6.167

9. Suppression of hypoglossal motoneurons during the carbachol-induced atonia of REM sleep is not caused by fast synaptic inhibition.

Authors: L Kubin; H Kimura; H Tojima; R O Davies; A I Pack
Journal: Brain Res Date: 1993-05-21 Impact factor: 3.252

10. An endogenous glutamatergic drive onto somatic motoneurons contributes to the stereotypical pattern of muscle tone across the sleep-wake cycle.

Authors: Christian Burgess; Diane Lai; Jerome Siegel; John Peever
Journal: J Neurosci Date: 2008-04-30 Impact factor: 6.167

7 in total

7. Evidence that adrenergic ventrolateral medullary cells are activated whereas precerebellar lateral reticular nucleus neurons are suppressed during REM sleep.

Authors: Georg M Stettner; Yanlin Lei; Kate Benincasa Herr; Leszek Kubin
Journal: PLoS One Date: 2013-04-22 Impact factor: 3.240

7 in total

Adventures and tribulations in the search for the mechanisms of the atonia of REM sleep.

1. Hypoglossal motoneurons are postsynaptically inhibited during carbachol-induced rapid eye movement sleep.

2. Noradrenergic, serotonergic and GABAergic antagonists injected together into the XII nucleus abolish the REM sleep-like depression of hypoglossal motoneuronal activity.

3. REM sleep-like atonia of hypoglossal (XII) motoneurons is caused by loss of noradrenergic and serotonergic inputs.

4. Disinhibition of perifornical hypothalamic neurones activates noradrenergic neurones and blocks pontine carbachol-induced REM sleep-like episodes in rats.

5. The postsynaptic inhibitory control of lumbar motoneurons during the atonia of active sleep: effect of strychnine on motoneuron properties.

6. Evidence that glycine mediates the postsynaptic potentials that inhibit lumbar motoneurons during the atonia of active sleep.

7. Behavioral state-specific inhibitory postsynaptic potentials impinge on cat lumbar motoneurons during active sleep.

8. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia.

9. Suppression of hypoglossal motoneurons during the carbachol-induced atonia of REM sleep is not caused by fast synaptic inhibition.

10. An endogenous glutamatergic drive onto somatic motoneurons contributes to the stereotypical pattern of muscle tone across the sleep-wake cycle.

1. Unraveling the mechanisms of REM sleep atonia.

2. Factual errors in Brooks and Peever's rebuttal to critiques.

3. Quantitative analysis of the excitability of hypoglossal motoneurons during natural sleep in the rat.

4. Activity of pontine A7 noradrenergic neurons is suppressed during REM sleep.

5. Medullary circuitry regulating rapid eye movement sleep and motor atonia.

Review 6. The anatomical, cellular and synaptic basis of motor atonia during rapid eye movement sleep.

7. Evidence that adrenergic ventrolateral medullary cells are activated whereas precerebellar lateral reticular nucleus neurons are suppressed during REM sleep.