| Literature DB >> 18954879 |
Min Lou1, Chao-Hui Jing, Magdy H Selim, Louis R Caplan, Mei-Ping Ding.
Abstract
Delayed post-anoxic encephalopathy has rarely been reported, and has most commonly been associated with carbon monoxide poisoning. The underlying pathophysiological mechanism is unknown. We describe a patient with a delayed neurologic decline after a good initial recovery from a hypoxic-ischemic event precipitated by acute blood loss. MRI revealed early globus pallidus necrosis followed by delayed substantia nigra damage and leukoencephalopathy. We suggest that anemia might play a role in this pattern of hypoxic-ischemic brain injury.Entities:
Mesh:
Year: 2008 PMID: 18954879 DOI: 10.1016/j.jns.2008.09.032
Source DB: PubMed Journal: J Neurol Sci ISSN: 0022-510X Impact factor: 3.181