Literature DB >> 18930060

Down-regulated in adenoma Cl/HCO3 exchanger couples with Na/H exchanger 3 for NaCl absorption in murine small intestine.

Nancy M Walker1, Janet E Simpson, Pei-Fen Yen, Ravinder K Gill, Elizabeth V Rigsby, Jennifer M Brazill, Pradeep K Dudeja, Clifford W Schweinfest, Lane L Clarke.   

Abstract

BACKGROUND & AIMS: Electroneutral NaCl absorption across small intestine contributes importantly to systemic fluid balance. Disturbances in this process occur in both obstructive and diarrheal diseases, eg, cystic fibrosis, secretory diarrhea. NaCl absorption involves coupling of Cl(-)/HCO(3)(-) exchanger(s) primarily with Na(+)/H(+) exchanger 3 (Nhe3) at the apical membrane of intestinal epithelia. Identity of the coupling Cl(-)/HCO(3)(-) exchanger(s) was investigated using mice with gene-targeted knockout (KO) of Cl(-)/HCO(3)(-) exchangers: Slc26a3, down-regulated in adenoma (Dra) or Slc26a6, putative anion transporter-1 (Pat-1).
METHODS: Intracellular pH (pH(i)) of intact jejunal villous epithelium was measured by ratiometric microfluoroscopy. Ussing chambers were used to measure transepithelial (22)Na(36)Cl flux across murine jejunum, a site of electroneutral NaCl absorption. Expression was estimated using immunofluorescence and quantitative polymerase chain reaction.
RESULTS: Basal pH(i) of DraKO epithelium, but not Pat-1KO epithelium, was alkaline, whereas pH(i) in the Nhe3KO was acidic relative to wild-type. Altered pH(i) was associated with robust Na(+)/H(+) and Cl(-)/HCO(3)(-) exchange activity in the DraKO and Nhe3KO villous epithelium, respectively. Contrary to genetic ablation, pharmacologic inhibition of Nhe3 in wild-type did not alter pH(i) but coordinately inhibited Dra. Flux studies revealed that Cl(-) absorption was essentially abolished (>80%) in the DraKO and little changed (<20%) in the Pat-1KO jejunum. Net Na(+) absorption was unaffected. Immunofluorescence demonstrated modest Dra expression in the jejunum relative to large intestine. Functional and expression studies did not indicate compensatory changes in relevant transporters.
CONCLUSIONS: These studies provide functional evidence that Dra is the major Cl(-)/HCO(3)(-) exchanger coupled with Nhe3 for electroneutral NaCl absorption across mammalian small intestine.

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Year:  2008        PMID: 18930060      PMCID: PMC2673535          DOI: 10.1053/j.gastro.2008.07.083

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  39 in total

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Journal:  EMBO J       Date:  2002-11-01       Impact factor: 11.598

2.  Identification of an apical Cl(-)/HCO3(-) exchanger in the small intestine.

Authors:  Zhaohui Wang; Snezana Petrovic; Elizabeth Mann; Manoocher Soleimani
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3.  Intestinal NaCl transport in NHE2 and NHE3 knockout mice.

Authors:  Lara R Gawenis; Xavier Stien; Gary E Shull; Patrick J Schultheis; Alison L Woo; Nancy M Walker; Lane L Clarke
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4.  Gastrointestinal distribution and kinetic characterization of the sodium-hydrogen exchanger isoform 8 (NHE8).

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5.  Lateral intercellular space volume as a determinant of CFTR-mediated anion secretion across small intestinal mucosa.

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Authors:  David B Mount; Michael F Romero
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9.  Molecular characterization of the murine Slc26a6 anion exchanger: functional comparison with Slc26a1.

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Journal:  Am J Physiol Renal Physiol       Date:  2002-10

10.  Acute regulation of the SLC26A3 congenital chloride diarrhoea anion exchanger (DRA) expressed in Xenopus oocytes.

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4.  Loss of downregulated in adenoma (DRA) impairs mucosal HCO3(-) secretion in murine ileocolonic inflammation.

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Review 6.  Recent advances in small bowel diseases: Part I.

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8.  Differential association of the Na+/H+ Exchanger Regulatory Factor (NHERF) family of adaptor proteins with the raft- and the non-raft brush border membrane fractions of NHE3.

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9.  Ion Transport Basis of Diarrhea in a Mouse Model of Adoptive T Cell Transfer Colitis.

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10.  Characterization of CFTR High Expresser cells in the intestine.

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