Literature DB >> 18847373

Differential recruitment of dendritic cells and monocytes to respiratory mucosal sites in children with influenza virus or respiratory syncytial virus infection.

Michelle A Gill1, Kristin Long, Theresa Kwon, Luz Muniz, Asuncion Mejias, John Connolly, Lonnie Roy, Jacques Banchereau, Octavio Ramilo.   

Abstract

BACKGROUND: Influenza virus and respiratory syncytial virus (RSV) are among the most common viruses causing infections of the lower respiratory tract in young children. Although there are important differences in the immunopathogenesis of these 2 viral pathogens, little is known about how they affect antigen-presenting cells in children with acute infections.
METHODS: To characterize the immune cells that are mobilized to the respiratory tract by influenza virus and RSV, we analyzed nasal wash and blood samples obtained from children hospitalized with acute respiratory infections.
RESULTS: Influenza virus and RSV mobilize immune cells, including myeloid dendritic cells (mDCs) and plasmacytoid dendritic cells (pDCs), to the nasal mucosa. Patients with influenza virus infection had greater numbers of mDCs, pDCs, and monocytes in nasal wash samples than did patients with RSV infection. The frequencies of respiratory tract and blood T cell subsets were not affected by infection with influenza virus or RSV. Monocyte chemoattractant protein-1 concentrations in nasal wash samples were significantly increased in patients with influenza virus infection but not in those with RSV infection. RANTES (regulated on activation, normally T cell expressed and secreted) concentrations were increased only in the blood of patients with influenza virus infection.
CONCLUSIONS: Infection with influenza virus or RSV mobilizes antigen-presenting cells to the respiratory tract. The differences in antigen-presenting cell numbers and cytokine concentrations suggest that there are distinctive, early immune responses to these 2 viruses.

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Year:  2008        PMID: 18847373      PMCID: PMC2696361          DOI: 10.1086/593018

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  34 in total

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Authors:  R M CHANOCK; R H PARROTT
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2.  The distinctive features of influenza virus infection of dendritic cells.

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4.  Respiratory syncytial virus in early life and risk of wheeze and allergy by age 13 years.

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Authors:  Roberto P Garofalo; Karen H Hintz; Vanessa Hill; John Patti; Pearay L Ogra; Robert C Welliver
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8.  Mobilization of plasmacytoid and myeloid dendritic cells to mucosal sites in children with respiratory syncytial virus and other viral respiratory infections.

Authors:  Michelle A Gill; A Karolina Palucka; Theresa Barton; Faryal Ghaffar; Hasan Jafri; Jacques Banchereau; Octavio Ramilo
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Authors:  Roberto P Garofalo; Karen H Hintz; Vanessa Hill; Pearay L Ogra; Robert C Welliver
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Authors:  R Malley; J DeVincenzo; O Ramilo; P H Dennehy; H C Meissner; W C Gruber; P J Sanchez; H Jafri; J Balsley; D Carlin; S Buckingham; L Vernacchio; D M Ambrosino
Journal:  J Infect Dis       Date:  1998-12       Impact factor: 5.226

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  45 in total

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3.  Aging impairs both primary and secondary RIG-I signaling for interferon induction in human monocytes.

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5.  Respiratory Syncytial Virus Genotypes, Host Immune Profiles, and Disease Severity in Young Children Hospitalized With Bronchiolitis.

Authors:  Rosa Rodriguez-Fernandez; Lorena I Tapia; Chin-Fen Yang; Juan Pablo Torres; Susana Chavez-Bueno; Carla Garcia; Lisa M Jaramillo; Melissa Moore-Clingenpeel; Hasan S Jafri; Mark E Peeples; Pedro A Piedra; Octavio Ramilo; Asuncion Mejias
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6.  Respiratory syncytial virus (RSV) RNA loads in peripheral blood correlates with disease severity in mice.

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8.  IgE cross-linking critically impairs human monocyte function by blocking phagocytosis.

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9.  Innate immune dysfunction is associated with enhanced disease severity in infants with severe respiratory syncytial virus bronchiolitis.

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10.  Respiratory syncytial virus-induced activation and migration of respiratory dendritic cells and subsequent antigen presentation in the lung-draining lymph node.

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