Literature DB >> 1884444

The long QT syndrome. Prospective longitudinal study of 328 families.

A J Moss1, P J Schwartz, R S Crampton, D Tzivoni, E H Locati, J MacCluer, W J Hall, L Weitkamp, G M Vincent, A Garson.   

Abstract

BACKGROUND: The Long QT Syndrome (LQTS) is an infrequently occurring familial disorder in which affected individuals have electrocardiographic QT interval prolongation and a propensity to ventricular tachyarrhythmic syncope and sudden death. We prospectively investigated the clinical characteristics and the long-term course of 3,343 individuals from 328 families in which one or more members were identified as affected with LQTS (QTc greater than 0.44 sec1/2). METHODS AND
RESULTS: The first member of a family to be identified with LQTS, the proband, was usually brought to medical attention because of a syncopal episode during childhood or teenage years. Probands (n = 328) were younger at first contact (age 21 +/- 15 years), more likely to be female (69%), and had a higher frequency of preenrollment syncope or cardiac arrest with resuscitation (80%), congenital deafness (7%), a resting heart rate less than 60 beats/min (31%), QTc greater than or equal to 0.50 sec1/2 (52%), and a history of ventricular tachyarrhythmia (47%) than other affected (n = 688) and unaffected (n = 1,004) family members. Arrhythmogenic syncope often occurred in association with acute physical, emotional, or auditory arousal. The syncopal episodes were frequently misinterpreted as a seizure disorder. By age 12 years, 50% of the probands had experienced at least one syncopal episode or death. The rates of postenrollment syncope (one or more episodes) and probable LQTS-related death (before age 50 years) for probands (n = 235; average follow-up 54 months per patient) were 5.0% per year and 0.9% per year, respectively; these event rates were considerably higher than those observed among affected and unaffected family members.
CONCLUSIONS: Among 232 probands and 1,264 family members with prospective follow-up, three factors made significant independent contributions to the risk of subsequent syncope or probable LQTS-related death before age 50 years, whichever occurred first (Cox hazard ratio; 95% confidence limits): 1) QTc (1.052; 1.017, 1.088), 2) history of cardiac event (3.1; 1.3, 7.2), and 3) heart rate (1.017; 1.004, 1.031). The findings from this prospective longitudinal study highlight the clinical features, risk factors, and course of LQTS.

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Year:  1991        PMID: 1884444     DOI: 10.1161/01.cir.84.3.1136

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  186 in total

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2.  New era for digital ECG: FDA requires digital ECG submission for tested drugs.

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3.  Variable expression of long QT syndrome among gene carriers from families with five different HERG mutations.

Authors:  Jesaia Benhorin; Arthur J Moss; Matthew Bak; Wojciech Zareba; Elizabeth S Kaufman; Batsheva Kerem; Jeffrey A Towbin; Silvia Priori; Robert S Kass; Bernard Attali; Arthur M Brown; Eckhard Ficker
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4.  Risk of recurrent cardiac events after onset of menopause in women with congenital long-QT syndrome types 1 and 2.

Authors:  Jonathan Buber; Jehu Mathew; Arthur J Moss; W Jackson Hall; Alon Barsheshet; Scott McNitt; Jennifer L Robinson; Wojciech Zareba; Michael J Ackerman; Elizabeth S Kaufman; David Luria; Michael Eldar; Jeffrey A Towbin; Michael Vincent; Ilan Goldenberg
Journal:  Circulation       Date:  2011-05-31       Impact factor: 29.690

5.  Long QT Syndromes.

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7.  Determinants of prolonged QT interval and their contribution to sudden death risk in coronary artery disease: the Oregon Sudden Unexpected Death Study.

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Review 9.  Long QT syndrome: novel insights into the mechanisms of cardiac arrhythmias.

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10.  Effect of phenylephrine provocation on dispersion of repolarization in congenital long QT syndrome.

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